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Among various photosensitizing dyes, 4, 4'-dimethyl 3, 3'-di-n-heptyl-8- {2-(4-methyl-3-n-heptylthiazole) }-2, 2'-dicarbocyanin diiodide (abb. NK19), even in an extremely low concentration, is known to inhibit the proliferation of bacteria and tissue culture cells (1, 2, 3). With respect to the mechanism of such inhibitory action no other property of this NKl9 is known except that it has a marked adsorptive property to protein (4). As a step toward the elucidation of the mode of biological effect, the author studied the effect of NK19 on the energy transfer reaction of Irat liver mitochondria, followed by comparison with the mode of actions of various other inhibitors of the oxidative phosphorylation (5). NK19. NKl9 can be prepared by letting 2, 4-dimethylthiazole heptyliodide react with ethylorthoformate in anhydrous acetic acid. We used NKI9, a product of Nihon Kanko Shikiso Research Laboratories. The molecular structure is as in the following and in its MeOH state it has maximum absorbancy at 590 m,a. For the use in experiment it was made into 1 mg/ml of MeOH and was stored in the dark until used.

A cell strain having low tumor-producing capacity was exposed in culture to 3'-methyl-N,N-dimethyl-4-aminoazobenzene (3'-Me-DAB) in the presence or absence of liver microsomes, and whether or not the cells will progress to those having high tumor-producing capacity was examined. When transplanted into rats, the cells treated with 3'-Me-DAB four (Exp-I) or thirteen times (Exp-II) formed larger tumors than untreated control cells, the latter treatment being more efficient in this regard. Furthermore, the tumors formed by the cells treated with 3'-Me-DAB in the presence of liver microsomes were considerably larger than those formed by the cells treated with 3'-Me-DAB alone. The subcutaneous tumors produced by the cells treated with 3'-Me-DAB with S-15 Mix showed poorly differentiated histology compared with those produced by control and 3'-Me-DAB-treated cells. The frequency of lung metastasis tended to increase by 3'-Me-DAB with S-15 Mix. The cells treated with 3'-Me-DAB in the presence or absence of liver microsomes differed from untreated control cells in vitro in some properties, including the size of aggregates in rotation culture, plating efficiency in liquid medium and morphology. These observations suggest that cell malignancy was promoted by 3'-Me-DAB alone as well as by 3'-Me-DAB in the presence of liver microsomes.

To investigate whether an association exists between vascular endothelial growth factor (VEGF) expression and tumor prognosis in primary lung carcinoma, we used immunohistochemical techniques to analyze microvessel density and VEGF expression in lung carcinoma tissue from 98 patients. Tissue had been fresh-frozen at the time of operation and preserved for more than 5 years. The results indicated that VEGF expression was positive for 50 of the 98 patients (51.0%), with 27 (27.6%) being weakly positive and 23 (23.5%) being strongly positive. The microvessel density in tissue showing weakly positive and strongly positive VEGF expression was significantly higher than that in VEGF-negative tumor tissue (P < 0.05: negative vs. weakly positive, P < 0.01: negative vs. strongly positive), we showed demonstrating that VEGF expression was significantly associated with intratumoral microvessel density. The 5-year survival rates were 8.7% for strongly VEGF-positive patients, 43.9% for weakly VEGF-positive patients and 79.2% for VEGF-negative patients, respectively (P < 0.01: negative vs. weakly positive or strongly positive). Furthermore, multivariate analysis employing multiple regression analysis indicated that VEGF expression correlates highly with the overall survival rates of patients with primary lung carcinoma. Two variables, N status and VEGF expression, were found to be significant prognostic factors (P < 0.01). The results of this study suggest that VEGF expression is associated with intratumoral microvessel density. VEGF expression may constitute important independent prognostic evidence that can help us in predicting the outcomes of patients with primary lung carcinomas.

Effects of antioxidants, such as superoxide dismutase, vitamin C, vitamin E, 4-(0-benzylphenoxy)-N-methylbutylamine hydrochloride (bifemelane), and selenite on survival of adult rat hepatocytes were examined under normoxic and hyperoxic conditions in serum-free primary culture. The tested antioxidants, except for vitamin C, significantly increased the survival rate of hepatocytes under the normoxic condition (under air). Thus, even the normoxic culture condition is hyperoxic for hepatocytes. Elevation of oxygen tension (40% O2) caused severe morphologic degeneration of hepatocytes and remarkable decrease in the survival rate of the cells. Addition of the antioxidants effectively protected hepatocytes from the morphologic degeneration, and significantly improved the survival of the cells under the hyperoxic condition. These findings indicate that the antioxidants can maintain the long-term survival of hepatocytes in serum-free primary culture.

Genetic variation of hepatitis C virus was assessed. We prepared RNA fractions from 21 patients' sera which were positive for hepatitis C virus RNA, synthesized their cDNAs, and amplified fragments, 406 base pairs, encoding a putative core protein, by polymerase chain reaction. One of them, N 15, was cloned and sequenced. N 15 showed 92.4% homology at the nucleotide level and 97.0% homology at the amino acid level compared with HC-J 1 which is the first isolated clone in Japan and similar to that isolated in USA. By restriction fragment length polymorphisms analysis, 14 out of 21 patients (66.7%) showed the same pattern as N 15. No patients showed the pattern of HC-J 1. We could not find a correlation between the genetic variation and clinical features of hepatitis C virus infection. These results indicate that the region, which encodes the core protein and is believed to be relatively conserved in hepatitis C virus genome, has several variations at the nucleotide level, and the major part of hepatitis C virus in Okayama district is different from HC-J 1 and the USA clone.

Aus dem Gesagten ergibt sich folgendes : 1) Nach Darreichung einer kleinen Menge von KBr kommt beim Kaninchen der Golgische Apparat der Leberzellen in Gestalt sehr zahlreicher feiner stabchen- oder fadenformiger Elemente zum Vorschein. Diese finden sich am dichtesten an den Gallenkapillaren und sind dort besonders fein, sodass man den Eindruck hat, als ob sie in feinere Kornchen zerfallen und in die Gallenkapillaren eindringen. Wenn man aber eine grossere Dosis des Mlittels gebraucht, so nehmen die Kornchen an Zahl ab, an Grbsse aber zu. Dabei ist ihre innige Beziehung zu den Gallenkapillaren nicht mehr erkennbar. Endlich verschwinden sie bei der hochgradigen Beschadigung der Zellfunktion ganz und gar. 2) Die Veranderung des Golgicshen Apparates in den Leberzellen, die durch Darreichung von KJ zustande kommt, stimmt mit dem oben Genannten fast uberein, abgesehen davon, dass hier die Grosse der Kornchen etwas grober ist. 3) Nach Darreichung von KC1 kann man ebenfalls einen fast ahnlichen Befund wie bei den vorigen Failen konstatieren, jedoch sieht man haufig anstatt des Golgischen Apparates eine grosse schwarze Masse, die sicher als Niederschlag anzusehen ist und besonders nach Darreichung einer grossen Menge von KCI auftritt. 4) Nach Darreichung von KN03 stellt sich der Golgsche Apparat in Gestalt von verhaltnismossig grbsseren Kornchen dar, die hauptsah lich in der Nahe der Gallenkapillaren vorhanden sind, wenn man sie auch uberall im ganzen Zelleib findet. 5) Nach Darreichung von Na2S04 verhalt sich der Golgische. Apparat wie irn Falle von KN03. Aus den oben erwahnten verschieclenen Befunden ergibt sich, dass der Golgische Apparat der Leberzellen nach der innerlichen Darreichung verschieclener Salze eine Veranderung erleidet, die vor allem je nach der Dosis des gebrauchten Mittels verschieden ist. Eine starke Beschadigung , der Zellfunktion durch Aufnahme einer grossen Dosis beeinflusst den Golgischen Apparat auf das schwerste, so sehr, dass er endlich vernichtet wird. Auch die Wirkung der genannten Mittel auf den Apparat ist je nach der Art der Salze mehr oder wenibger verschieden. Besonders auffallend ist in dieser Beziehung der Unterschied zwischen Halogen- und anderen Verbindungen. Die ersteren lassen den Golgischen Apparat in feine Kornchen zerfalle,n und in die Gallenkapillaren eindringen, was nach Darreichusg von KN03 oder Na2S04 nicht der Fall ist. Im allgemeinen hat das einwertige Anion die Wirkungg den Zelleib zu lockern, indem es die Dispersitat der Kolloide steigert. Vor allem gilt dies fur Halogenanionen. Was die Verschiebungsgeschwindigkeit und Penetrationskraft dieser Anionen anbelangt, so gibt, es keinen deutlichen Unterschied zwischen Br', J' und Cl', jedoch scheint es, dass Br' in dieser Beziehung den zwei andern etwas voransteht. Die Halogenionen im Blute scheiden sich schnell in die Galle, wobei sie die Gallensekretion befordern. Hisiologisch kann man diese Tatsac.he dufch die oberwahnte Veranderung des Golgischen Apparates in den Leberzellen nachweisen. N'03' u.nd S04" haben nur eine geringe Wanderungsgeschwincligkeit und Eindringung'skraft. S04" entlwasse.rt sogar die Zellen, indem es lange Zeit ausserhalb der Zelle bleibt. Was die Gallensekretion anbelangt, so uben N03' und S04" keinen bedeutenden Einfluss auf sie aus, was auch histologisch durch den Befund des Golgischen Apparates in den Leberzellen nach Darreichung von KN03 oder Na2S04 erkennbar ist.

In this study, we investigated the effects of both 25 and 50 mg daily doses of rofecoxib on the endothelial functions of patients with coronary artery disease (CAD). For this purpose, 34 patients with documented severe CAD and who were under aspirin treatment (300 mg/day) were randomized to receive 4 weeks of treatment with a placebo (n = 10, group I), rofecoxib 25 mg/day (n = 12, group II), and rofecoxib 50 mg/day (n = 12, group III). Brachial artery vasodilator responses were measured in order to evaluate endothelial function. The percentage of change in endothelial-dependent vasodilation in groups I, II, and III were similar at the baseline level and showed no significant change after treatment (6.2+/-3.9% vs. 5.9+/-3.1% and 5.8+/-3.3% vs. 5.6+/-3.8% and 6.1+/-4.5% vs. 5.8+/-4.1%, respectively; P > 0.05). Compared with the baseline, endothelium-independent vasodilatation, as assessed by nitroglycerine (NTG), remained unchanged after the treatment period (11.2+/-6.9% vs. 10.3+/-7.1% and 11.2+/-6.3% vs. 9.9+/-5.1% and 9.5+/-4.9% and 8.8+/-4.6%, respectively; P> 0.05). Treatment with both doses also showed no significant effects on high-sensitivity C-reactive protein (hs-CRP) levels and resting arterial diameters (P > 0.05). In conclusion, 4 weeks of treatment with standard and high doses of rofecoxib showed no significant effects on either endothelial-dependent or independent vasodilator response or plasma hs-CRP levels in patients with severe CAD taking concomitant aspirin.

Changes in brain vascularity in adult rats during adaptation to chronic normobaric hypoxia with or without elevated CO(2) were morphometrically investigated. Immunohistochemistry with anti-rat endothelial cell antigen (RECA-1) antibody was carried out for the vascular analysis. After the rats were subjected to hypoxia for 2 to 8 weeks (wks)(10 percent O(2) in N(2)), the total area of blood vessels was measured in 6 brain regions. After 2 wks of hypoxia, the blood vessel area was found to be significantly increased in the frontal cortex, striatum, hippocampus, thalamus, cerebellum, and medulla oblongata, by 44% , 96% , 65% , 50% , 102% and 97% , respectively. The ratio of large vessels with an area > 500 micro m(2) was also increased in all brain regions. Hypoxic adaptation in brain vascularity did not change during 8 wks of hypoxia, and the hypoxia-induced levels measured in the vasculature returned to control levels 2 wks after the termination of hypoxia in areas of the brain other than the cortex and thalamus. In addition, hypoxia-induced changes in terms of the total vascular area and vessel size distribution were significantly inhibited by the elevation in CO(2), whereas chronic hypercapnia without hypoxia had no effect on brain vascularity. These findings suggested that adaptations in brain vascularity in response to hypoxia are rapidly induced, and there are regional differences in the reversibility of such vascular changes. Carbon dioxide is a potent suppressor of hypoxia-induced vascular changes, and may play an important role in vascular remodeling during the process of adaptation to chronic hypoxia.

Ferric nitrilotriacetate (Fe3+-NTA) solution showed maximum absorbance at pH 7.5. The iron was in ferric high-spin state and coordinated octahedrally with a relatively symmetric structure and also probably pentagonally. A spin trapping technique employing 5,5-dimethyl-1-pyrroline-N-oxide (DMPO) yielded a DMPO spin adduct of unknown radical with three doublets (DMPO-Z) and a simple nitroxide radical (Y-NO.) in serum from rats injected intraperitoneally with Fe3+-NTA. When the Fe3+-NTA solution was diluted 500-fold with 50 mM NTA solution, DMPO-Z, Y-NO. and an additional signal, DMPO-OH were observed. The DMPO-Z signal was suppressed by a decrease in oxygen tension, alpha-tocopherol and 3-tert-butyl-4-hydroxy-anisole (BHA). The DMPO-OH signal was suppressed in the presence of ethanol and catalase. Fe2+-NTA solution hardly produced DMPO spin adducts. The Fe3+-NTA solution produced a strong DMPO-OH signal in the presence of H2O2. Rose Bengal solution, a singlet oxygen generating system, produced the same DMPO adducts. Fe3+-NTA reacted with oxygen in solution. The oxygen was activated and might be similar to singlet molecular oxygen. In the presence of H2O2, the Fe3+-NTA solution generated a hydroxyl radical. Fe3+-NTA itself generated free radicals, but Fe2+-NTA did not.

To investigate the involvement of actin filaments in concanavalin A (Con A)-induced cap formation and cytochalasin B (CB)-induced zeiotic knob migration, the distribution of F-actin was studied in Con A-treated and CB-treated Ehrlich ascites tumor cells (EATC) by fluorescence microscopy using heavy meromyosin conjugated with a fluorescent dye, N-(7-dimethylamino-4-methylcoumarinyl) maleimide, (DACM-HMM). In non-treated cells, the diffuse fluorescence of DACM-HMM was observed in the cytoplasm, particularly intensely under the plasma membrane and around the nucleus. In Con A- and CB-treated cells, the fluorescence was seen at Con A-induced-capped and CB-induced-knob-accumulated regions. This fluorescence was more intense in CB-treated cells. To study the actin filaments in these fluorescent regions more clearly, the soluble components of the cells were eliminated by treatment with Triton X-100 or saponin solution containing a low concentration of glutaraldehyde, and the detergent-treated and saponin-treated cells were observed under a transmission electron microscope. Concentrated actin filaments were observed directly beneath the Con A-induced capping area and CB-induced zeiotic knob-accumulation area. The area of concentrated actin filaments appeared to correspond to the electron dense area observed in the identical region in the cells fixed without detergent treatment. More actin filaments were observed in CB-treated cells than in Con A-treated ones.

Caryophyllaeidae Claus, 1879 1. Glaridacris limnodrili Yamaguti, 1934 Bothriocephalidae Blanchard, 1849 2. Bothriocephalus fluviatilis n. sp. 3. B. lateolabracis n. sp. 4. B. branchiostegi n. sp. 5. B. acheilognathi Yamaguti, 1934 6. B. brotulae n. sp. 7. B. apogonis n. sp. 8. Oncodiscus sauridae Yamaguti, 1934 9. Glossobothrium nipponicum n. g., n. sp. Amphicotylidae Ariola, 1899 10. Amphicotyle quinquarii n. sp. 11. Eubothrioides lamellatus n. g., n. sp. Phyllobothriidae Braun, 1900 12. Phyllobothrium triacis n. sp. 13. P. filiforme n. sp. 14. P. serratum n. sp. 15. P. laciniatum (Linton, 1889) 16. P. loculatum n. sp. 17. P. squali n. sp. 18. P. lactuca van Beneden, 1850 19. Anthobothrium rajae n. sp. 20. A. pteroplateae n. sp. 21. A. bifidum n. sp. 22. A. parvum Stossich, 1895 23. Orygmatobothrium musteli (van Beneden, 1850) 24. O. versatile Die3ing, 1854 25. Monorygma megacotyla n. sp. 26. Pithophorus vulpeculae n. sp. 27. Echeneibothrium bifidum n. sp. 28. E. tobijei Yamaguti, 1934 29. Marsupiobothrium alopias n. g ., n. sp. 30. Dinobothrium spinulosum n. sp. 31. Gastrolecithus planus (Linton, 1922) n. g. Onchobothriidae Braun, 1900 32. Acanthobothrium triads n. sp. 33. A. micracantha n. sp. 34. A. latum n. sp. 35. A. gracile n. sp. 36. A. dasybati Yamaguti, 1934 37. A. ijimai Yoshida, 1917 38. A. grandiceps n. sp. 39. Calliobothrium verticillatum (Rud., 1819) van Bened., 1850 40. Platybothrium auriculatum n. sp. 41. P. musteli n. sp. Aberrant Tetraphyllidea 42. Pelichnibothrium speciosum Montic., 1889 43. Discobothrium japonicum Yamaguti, 1934 Tentaculariidae Poche, 1926 44. Nyelinia manazo n. sp. 45. N. sphyrnae n. sp. Floricipitidae Dollfus, 1929 46. Floriceps uncinatus (Linton, 1924) Tetrarhynchidean larvae 47. Pintneriella musculicola Yamaguti. 1934 48. Microbothriorhynchus coelorhynchi n. g., n. sp. 49. Oncomegas wageneri (Linton, 1890) 50. Pterobothrium chaeturichthydis n. sp. 51. P. hira n. sp. 52. Callotetrarhynchus speciosus (Linton, 1897) 53. Symbothriorhynchus uranoscopi n. g., n. sp. 54. Nybelinia anguillae n. sp. 55. N. nipponica n. sp. 56. Otobothrium dipsacum Linton, 1897

I. Dactylogyridae Bychowsky, 1933 1. Ancyrocephalus macrogaster n. sp. 2. Ancyrocephalus bilobatus n. sp. 3. Ancyrocephalus spinicirrus n. sp. 4. Ancyrocephalus platycephali n. sp. 5. Haliotrema alatum Yamaguti, 1942 6. Haliotrema lutiani n. sp. 7. Haliotrema caesionis n. sp. 8. Haliotrema upenei n. sp. 9. Metahaliotrema scatophagi n. g., n. sp. 10. Metahaliotrema arii n. sp. 11. Pseudohaliotrema (Pseudohaliotrema) sphincteroporus n. g., n. sp. 12. Pseudohaliotrema (Pseudohaliotrema) sigani n. sp. 13. Pseudohaliotrema (Pseudohaliotrematoides) fusiforme n. subg., n. sp. 14. Hamatopeduncularia arii n. g., n. sp. 15. Diplectanum serrani n. sp. 16. Pseudolamellodiscus sphyraenae n. g., n. sp. 17. Lamellodiscus flexuosus n. sp. 18. Lamellodiscus convolutus n. sp. 19. Lamellodiscus difficilis n. sp. 20. Lamellodiscus duplicostatus n. sp. 21. Diplectanocotyla gracilis n. g., n. sp. II. Capsalidae Baird, 1853 22. Benedenia synagris n. sp. III. Mazocraeidae Price, 1936 23. Kuhnia scombri (Kuhn, 1829) Sproston, 1945 24. Kuhnia otolithis n. sp. IV. Discocotylidae Price, 1936 25. Allodiscocotyla chorinemi n. g., n. sp. 26. Vallisia chorinemi n. sp. 27. Protomicrocotyle celebesensis n. sp. V. Microcotylidae Taschenberg, 1879 28. Metamicrocotyla bora n. g., n. sp. 29. Metamicrocotyla filiformis n. sp. 30. Heteromicrocotyla carangis n. g., n. sp. 31. Gotocotyla mesercei n. sp.

Bucephalidae Poche, 1907 1. Prosorhynchus longicollis n. sp. 2. Rhipidocotyle khalili Nagaty, 1937 Allocreadiidae Stossich, 1904 3. Helicometra epinepheli Yamaguti, 1934 4. Opeehona scombri Yamaguti, 1938 5. Pseudopecoeloides tenuis Yamaguti, 1940 Schistorchiidae Yamaguti, 1942 6. Schistorchis sigani Yamaguti, 1942 7. Apocreadium synagris n. sp. Fellodistomidae Nicoll. 1913 8. Symmetrovesicula ehaetodontis Yamaguti, 1938 Monorchiidae Odhner, 1911 9. Lasiotocus lethrini n. sp. Heterophyidae Odhner, 1911 10. Paracryptogonimus aeanthostomus Yamaguti, 1934 Gyliauchenidae Ozaki, 1933 11. Gyliauchen nahaensis Ozaki, 1937 12. Gyliauehen papillatus (Goto et Matsudaira, 1918) Hemiuridae Luhe, 1901 13. Parahemiurus clupeae n. sp. 14. Aphanurus harengulae Yamaguti, 1938 15. Aphanurus dorosomatis n. sp. 16. Aponurus laguncula Looss, 1907 17. Aponurus synagris n. sp. 18. Lecithochirium priacanthi n. sp. 19. Lecithochirium longicaudatum n. sp. 20. Lecithocladium parviovum n. sp. 21. Lecithocladium megalaspis n. sp. 22. Lecithocladium angustiovum n. sp. 23. Lecithocladium scombri n. sp. 24. Tubulovesicula angusticauda (Nicoli, 1915) 25. Magnacetabulum leiognathi n. sp. 26. Hysterolecitha nahaensis Yamaguti, 1942 27. Hysterolecithoides epinepheli Yamaguti, 1934 28. Lecithaster stellatus Looss, 1907 Angiodictyidae Looss, 1902 29. Hexangium sigani Goto et Ozaki, 1929 Didymozoidae Poche, 1907 30. Didymozoon spirale Yamaguti, 1938 31. Didymozoon brevicolle Yamaguti, 1938 32. Unitubulotestis carangis n. g., n. sp.

I. Nematodes of fishes 1. Rhabdochona amago Yamaguti, 1935 II. Nematodes of frogs 2. Cosmocerca japonica Yamaguti, 1938 3. Cosmocercoides pulcher Wilkie, 1930 4. Oswaldocruzia bialata (Molin, 1860) 5. Rhabdias montana n. sp. III. Nematodes of mammals 6. Protospirura muris (Gmelin, 1890) 7. Heterakis spumosa Schneider, 1866 8. Longistriata wolgaensis Schulz, 1926 IV. Acanthocephala of frogs 9. Acanthocephalus lucidus van Cleave, 1925

I. Trematodes of reptiles 1. Cyathocotyle crocodili n. sp. 2. Pseudoneodiplostomum (Pseudoneodiplostomoides) crocodili n. subg. n. sp. 3. Acanthostomum crocodili n. sp. II. Trematodes of birds 4. Plagiorchis maculosus (Rud., 1ε02) 5. Echinochasmus bagulai Verma, 1935

Anoplocephalidae Kholodk., 1902 1. Oochoristica celebesensis n. sp. Dilepididae Fuhrmann, 1907 2. Ophiovalipora micracantha n. sp. Proteocephalidae La Rue, 1911 3. Acanthotaenia shipleyi von Linstow, 1903

Onchobothriidae Braun, 1900 1. Balanobothrium tenax Hornell, 1912 2. Balanobothrium stegostomatis n. sp. 3. Phoreiobothrium sp. Hornelliellidae n. fam. 4. Hornelliella annandalei (Hornell, 1912) n. g. Austramphilinidae Johnston, 1931 5. Gyrometra albotaenia n. g., n. sp.

Lecithodendriidae Odhner, 1910 1. Phaneropsolus simiae n. sp. Heterophyidae Odhner, 1914 2. Galactosomum canis n. sp. Paramphistomidae Fischoeder, 1901 3. Explanatum explanatum (Creplin, 1857) 4. Paramphistomum cervi (Schrank, 1790) 5. Calicophoron cauliorchis (Stiles et Goldberger, 1900) 6. Ceylonocotyle εcoliocoelium (Fischoeder, 1901) 7. Fischoederius elongatus (Poirier, 1883) Fasciolidae Railliet, 1895 8. Fasciola hepatica Linne, 1758

Echinorhynchidae Cobbold, 1879 1. Acanthocephalus bufonis (Shipley, 1903) Rhadinorhynchidae Travassos, 1923 2. Rhadinorhynchus celebesensis n. sp. 3. Filisoma indicum van Cleave, 1928 Quadrigyridae van Cleave, 1920 4. Pallisentis gaboes (MacCallum, 1918) van Cleave. 1928 Gigantorhynchidae Hamann, 1892 5. Empodius sp. Neoechinorhynchidae Hamann, 1892 6. Neoechinorhynchus longilemniscus n. sp.

I. Trematodes of mammals 1. Brachylaemus tokudai n. sp. 2. Acanthatrium ovatum Yamaguti, 1939 II. Cestodes of frogs 3. Baerietta montana n. sp. 4. Baerietta claviformis n. sp. III. Cestodes of· birds 5. Choanotaenia barbara Meggitt, 1926 IV. Cestodes of mammals 6. Hymenolepis apodemi n. sp.