検索結果 14881 件
| JaLCDOI | 10.18926/AMO/55588 |
|---|---|
| フルテキストURL | 71_6_513.pdf |
| 著者 | Sawada, Shigeki| Sugimoto, Ryujiro| Ueno, Tsuyoshi| Yamashita, Motohiro| |
| 抄録 | We evaluated the feasibility of maintenance treatment using UFT (a combination of tegafur and uracil) after adjuvant platinum-based chemotherapy in patients with resected lung cancer. A prospective feasibility trial was conducted. Between 2010 and 2014, UFT was administered for 2 years sequentially after platinum-based adjuvant chemotherapy in 24 patients with resected Stage IIA-IIIA non-small cell lung cancer. The safety of UFT and the rate of treatment completion were then evaluated. The prior platinum-based chemotherapy regimens consisted of cisplatin+vinorelbine in 16 patients, carboplatin+paclitaxel in 5 and carboplatin+S-1 in one. During the subsequent UFT administration, a total of 3 patients required a dose reduction because of Grade 1 blood-stained sputum, Grade 2 numbness, and Grade 2 constipation, in one patient each. Eleven patients underwent the planned 2-year UFT administration, but 12 patients could not because of the recurrence of lung cancer in 5 patients, metachronous malignancy in one, and toxicities in 6. The completion rate for UFT administration was 64.7% (11/17). The most common type of toxicity was gastrointestinal toxicities. All of the toxicities were grade 1 or 2, and no severe toxicities were observed. UFT treatment after platinum-based chemotherapy was revealed to be feasible. |
| キーワード | UFT adjuvant chemotherapy lung cancer resection |
| Amo Type | Original Article |
| 出版物タイトル | Acta Medica Okayama |
| 発行日 | 2017-12 |
| 巻 | 71巻 |
| 号 | 6号 |
| 出版者 | Okayama University Medical School |
| 開始ページ | 513 |
| 終了ページ | 518 |
| ISSN | 0386-300X |
| NCID | AA00508441 |
| 資料タイプ | 学術雑誌論文 |
| 言語 | 英語 |
| 著作権者 | CopyrightⒸ 2017 by Okayama University Medical School |
| 論文のバージョン | publisher |
| 査読 | 有り |
| PubMed ID | 29276224 |
| JaLCDOI | 10.18926/AMO/55587 |
|---|---|
| フルテキストURL | 71_6_505.pdf |
| 著者 | Honda, Yoshihiro| Takigawa, Nagio| Ichihara, Eiki| Ninomiya, Takashi| Kubo, Toshio| Ochi, Nobuaki| Yasugi, Masayuki| Murakami, Toshi| Yamane, Hiromichi| Tanimoto, Mitsune| Kiura, Katsuyuki| |
| 抄録 | (−)-Epigallocatechin-3-gallate (EGCG) has been shown to bind to several receptors including epidermal growth factor receptor (EGFR). EGFR tyrosine kinase inhibitors and anaplastic lymphoma kinase (ALK) inhibitors are effective for non-small cell lung cancers harboring activating EGFR mutations and ALK or c-ros oncogene 1 (ROS1) fusion genes, respectively. We investigated the effects of EGCG on EGFR- or fusion gene-driven lung cancer cells such as PC-9, RPC-9, H1975, H2228 and HCC78. The five cell lines had similar sensitivity to EGCG. Phosphorylated (p)EGFR, pAkt and pErk in PC-9, RPC-9 and H1975 cells were suppressed by EGCG (50 or 100 μM). EGCG also inhibited pALK in H2228, pROS1 in HCC78, and pErk and pAkt in both cell lines. All the xenograft tumors established using the 5 cell lines in EGCG-treated groups were significantly smaller than the tumors in the vehicle-treated groups. The numbers of tumor blood vessels of xenograft tissues in EGCG-treated mice were significantly lower than those in vehicle-treated mice. In conclusion, EGCG may be effective for EGFR-driven lung tumors irrespective of the presence of T790M, and for ALK or ROS1 fusion gene-driven lung tumors. |
| キーワード | epigallocatechin-3-gallate lung cancer EGFR ALK ROS1 |
| Amo Type | Original Article |
| 出版物タイトル | Acta Medica Okayama |
| 発行日 | 2017-12 |
| 巻 | 71巻 |
| 号 | 6号 |
| 出版者 | Okayama University Medical School |
| 開始ページ | 505 |
| 終了ページ | 512 |
| ISSN | 0386-300X |
| NCID | AA00508441 |
| 資料タイプ | 学術雑誌論文 |
| 言語 | 英語 |
| 著作権者 | CopyrightⒸ 2017 by Okayama University Medical School |
| 論文のバージョン | publisher |
| 査読 | 有り |
| PubMed ID | 29276223 |
| JaLCDOI | 10.18926/AMO/55586 |
|---|---|
| フルテキストURL | 71_6_493.pdf |
| 著者 | Aoe, Michinori| Ueno-Iio, Tomoe| Shibakura, Misako| Shinohata, Ryoko| Usui, Shinichi| Arao, Yujiro| Ikeda, Satoru| Miyahara, Nobuaki| Tanimoto, Mitsune| Kataoka, Mikio| |
| 抄録 | Lavender essential oil (Lvn) has anti-inflammatory effects in an ovalbumin-sensitized murine model of asthma, and inhibits inflammatory cell infiltration into the lungs. The anti-inflammatory effects of Lvn on cell adhesion molecules are not clear. Here we evaluated the effects of Lvn and its main constituents, linalyl acetate (LA) and linalool (LO), on the expression of tumor necrosis factor-alpha (TNF-α)-induced cell adhesion molecules in murine brain endothelial bEnd.3 cells and human umbilical vein endothelial cells (HUVECs). The bEnd.3 cells were treated with Lvn, LA, or LO and subsequently stimulated with TNF-α. The mRNA expression levels of cell adhesion molecules were detected using RT-PCR. E-selectin and P-selectin protein and phosphorylated-NF-κB p65 were detected by western blotting. The effects of Lvn on HUVECs were measured by RT-PCR. In bEnd.3 cells, Lvn and LA suppressed TNF-α-induced E-selectin, P-selectin, vascular cell adhesion molecule-1, intercellular adhesion molecule-1, and phosphorylated-NF-κB p65 in the nucleus; LO did not suppress P-selectin or phosphorylated-NF-κB p65. Lvn inhibited TNF-α-induced E-selectin mRNA in HUVECs. These results indicate that Lvn and LA inhibit TNF-α-induced cell adhesion molecules in endothelial cells through the suppression of NF-κB activation. Consequently, Lvn or other essential oils including LA may be useful as alternative anti-inflammatory medicines. |
| キーワード | lavender essential oil linalyl acetate inflammation cell adhesion molecule NF-κB |
| Amo Type | Original Article |
| 出版物タイトル | Acta Medica Okayama |
| 発行日 | 2017-12 |
| 巻 | 71巻 |
| 号 | 6号 |
| 出版者 | Okayama University Medical School |
| 開始ページ | 493 |
| 終了ページ | 503 |
| ISSN | 0386-300X |
| NCID | AA00508441 |
| 資料タイプ | 学術雑誌論文 |
| 言語 | 英語 |
| 著作権者 | CopyrightⒸ 2017 by Okayama University Medical School |
| 論文のバージョン | publisher |
| 査読 | 有り |
| PubMed ID | 29276222 |
| JaLCDOI | 10.18926/AMO/55585 |
|---|---|
| フルテキストURL | 71_6_485.pdf |
| 著者 | Okano, Ayaka| Masuhara, Shun| Ota, Sonoka| Motegi, Chie| Takabayashi, Noriko| Ogino, Tetsuya| |
| 抄録 | We examined postprandial body positions’ effects on gastrointestinal motility, the autonomic nervous system and subjective comfort, i.e., whether lowering the head after a meal is beneficial for gastrointestinal motility and the prevention of pressure ulcer. We examined 10 healthy subjects and compared 3 body positions: (1) Seated upright. (2) Lying on a bed with the head at 60° and knees up by 20° (60° position). (3) Identical to (2) until post-meal; the head was then lowered to 30° (60°-30° position). Gastrointestinal motility was assessed as gastrointestinal sounds measured by sound-editing software. Digital plethysmography assessed autonomic nerve function as heart rate variability. The pressure ulcer risk was estimated as subjective comfort/discomfort using a visual analog scale. Gastrointestinal sounds increased post-meal. The 60°-30° position showed the highest number of sounds and longest cumulative sound duration. Post-meal, sympathetic activation was suggested in the 60° position, whereas vagal activity was relatively preserved in the 60°-30° position. The 60°-30° position was the most comfortable, and the 60° position was least comfortable. Lowering the head after a meal is beneficial to augment gastrointestinal motility and decrease the pressure ulcer risk. The 60° head-up position increases the pressure ulcer risk. |
| キーワード | gastrointestinal sound body position autonomic nerve pressure ulcer patient care |
| Amo Type | Original Article |
| 出版物タイトル | Acta Medica Okayama |
| 発行日 | 2017-12 |
| 巻 | 71巻 |
| 号 | 6号 |
| 出版者 | Okayama University Medical School |
| 開始ページ | 485 |
| 終了ページ | 491 |
| ISSN | 0386-300X |
| NCID | AA00508441 |
| 資料タイプ | 学術雑誌論文 |
| 言語 | 英語 |
| 著作権者 | CopyrightⒸ 2017 by Okayama University Medical School |
| 論文のバージョン | publisher |
| 査読 | 有り |
| PubMed ID | 29276221 |
| JaLCDOI | 10.18926/AMO/55584 |
|---|---|
| フルテキストURL | 71_6_475.pdf |
| 著者 | Takei, Daisuke| Harada, Keita| Takashima, Shiho| Inokuchi, Toshihiro| Nakarai, Asuka| Sugihara, Yusaku| Takahara, Masanobu| Hiraoka, Sakiko| Okada, Hiroyuki| |
| 抄録 | Several reports discussed colonoscopic surveillance after polypectomy and endoscopic mucosal resection (EMR) for colorectal polyps, but only a few reports focused on prognostic analyses, and none involved metachronous neoplasia after colorectal endoscopic submucosal dissection (ESD). We conducted the present study to assess the risk of adenoma recurrence requiring endoscopic treatment, and to establish appropriate post-ESD colonoscopic surveillance. We enrolled 116 patients who had undergone colorectal ESD at Okayama University Hospital between February 2008 and July 2014 and had been followed-up >12 months. We retrospectively analyzed clinicopathological features of 101 lesions from 101 patients. Metachronous adenomas were detected in 21 cases (20.8%). We divided the patients into 2 groups according to the occurrence of metachronous adenomas. Our comparison of clinicopathological characteristics between these groups showed that in the metachronous adenomas group the number of synchronous adenomas at index colonoscopy was high and the rate of laterally spreading tumor-nongranular (LST-NG) was higher. A multivariate analysis indicated that the number of synchronous adenomas was significantly associated with metachronous adenomas (HR: 2.54, 95%CI: 1.04-6.52, p<0.05). The colonoscopic surveillance planning after colorectal ESD should be more meticulous for patients with more synchronous adenomas. |
| キーワード | endoscopic submucosal dissection laterally spreading tumor metachronous recurrence local recurrence post-ESD colonoscopic surveillance |
| Amo Type | Original Article |
| 出版物タイトル | Acta Medica Okayama |
| 発行日 | 2017-12 |
| 巻 | 71巻 |
| 号 | 6号 |
| 出版者 | Okayama University Medical School |
| 開始ページ | 475 |
| 終了ページ | 483 |
| ISSN | 0386-300X |
| NCID | AA00508441 |
| 資料タイプ | 学術雑誌論文 |
| 言語 | 英語 |
| 著作権者 | CopyrightⒸ 2017 by Okayama University Medical School |
| 論文のバージョン | publisher |
| 査読 | 有り |
| PubMed ID | 29276220 |
| JaLCDOI | 10.18926/AMO/55583 |
|---|---|
| フルテキストURL | 71_6_467.pdf |
| 著者 | Obata, Kengo| Yumoto, Tetsuya| Fuke, Soichiro| Tsukahara, Kohei| Naito, Hiromichi| Iida, Atsuyoshi| Takahashi, Tetsuya| Ujike, Yoshihito| Nakao, Atsunori| |
| 抄録 | Early mobilization is advocated to prevent intensive care unit-acquired physical weakness, but the patient's workload and its changes in response to body position changes have not been established. We used indirect calorimetry to determine the energy expenditure (EE) in response to body position changes, and we assessed EE's correlation with respiratory parameters in healthy volunteers: 8 males and 8 females, mean age 23.4±1.3 years. The subjects started in the resting supine position followed by a 30° head-up position, a 60° head-up position, an upright sitting position, a standing position, and the resting supine position. EE was determined in real time by indirect calorimetry monitoring the subject’s respiratory rate, tidal volume (VT), and minute volume (MV). The highest values were observed immediately after the subjects transitioned from standing to supine, and this was significantly higher compared to the original supine position (1,450±285 vs. 2,004±519 kcal/day, p<0.01). Moderate correlations were observed between VT and EE (r=0.609, p<0.001) and between MV and EE (r=0.576, p<0.001). Increasing VT or MV indicates an increasing patient workload during mobilization. Monitoring these parameters may contribute to safe rehabilitation. Further studies should assess EE in critically ill patients. |
| キーワード | early mobilization energy expenditure indirect calorimetry rehabilitation body position |
| Amo Type | Original Article |
| 出版物タイトル | Acta Medica Okayama |
| 発行日 | 2017-12 |
| 巻 | 71巻 |
| 号 | 6号 |
| 出版者 | Okayama University Medical School |
| 開始ページ | 467 |
| 終了ページ | 473 |
| ISSN | 0386-300X |
| NCID | AA00508441 |
| 資料タイプ | 学術雑誌論文 |
| 言語 | 英語 |
| 著作権者 | CopyrightⒸ 2017 by Okayama University Medical School |
| 論文のバージョン | publisher |
| 査読 | 有り |
| PubMed ID | 29276219 |
| JaLCDOI | 10.18926/AMO/55582 |
|---|---|
| フルテキストURL | 71_6_459.pdf |
| 著者 | Sakaguchi, Masakiyo| Kinoshita, Rie| Endy Widya Putranto| I Made Winarsa Ruma| I Wayan Sumardika| Youyi, Chen| Tomonobu, Naoko| Yamamoto, Ken-ichi| Murata, Hitoshi| |
| 抄録 | The receptor for advanced glycation end products (RAGE) is involved in inflammatory pathogenesis. It functions as a receptor to multiple ligands such as AGEs, HMGB1 and S100 proteins, activating multiple intracellular signaling pathways with each ligand binding. The molecular events by which ligand-activated RAGE controls diverse signaling are not well understood, but some progress was made recently. Accumulating evidence revealed that RAGE has multiple binding partners within the cytoplasm and on the plasma membrane. It was first pointed out in 2008 that RAGE’s cytoplasmic tail is able to recruit Diaphanous-1 (Dia-1), resulting in the acquisition of increased cellular motility through Rac1/Cdc42 activation. We also observed that within the cytosol, RAGE’s cytoplasmic tail behaves similarly to a Toll-like receptor (TLR4)-TIR domain, interacting with TIRAP and MyD88 adaptor molecules that in turn activate multiple downstream signals. Subsequent studies demonstrated the presence of an alternative adaptor molecule, DAP10, on the plasma membrane. The coupling of RAGE with DAP10 is critical for enhancing the RAGE-mediated survival signal. Interestingly, RAGE interaction on the membrane was not restricted to DAP10 alone. The chemotactic G-protein-coupled receptors (GPCRs) formyl peptide receptors1 and 2 (FPR1 and FPR2) also interacted with RAGE on the plasma membrane. Binding interaction between leukotriene B4 receptor 1 (BLT1) and RAGE was also demonstrated. All of the interactions affected the RAGE signal polarity. These findings indicate that functional interactions between RAGE and various molecules within the cytoplasmic area or on the membrane area coordinately regulate multiple ligand-mediated RAGE responses, leading to typical cellular phenotypes in several pathological settings. Here we review RAGE’s signaling diversity, to contribute to the understanding of the elaborate functions of RAGE in physiological and pathological contexts. |
| キーワード | receptor for advanced glycation end products RAGE adaptor protein signal transduction inflammatory pathogenesis |
| Amo Type | Review |
| 出版物タイトル | Acta Medica Okayama |
| 発行日 | 2017-12 |
| 巻 | 71巻 |
| 号 | 6号 |
| 出版者 | Okayama University Medical School |
| 開始ページ | 459 |
| 終了ページ | 465 |
| ISSN | 0386-300X |
| NCID | AA00508441 |
| 資料タイプ | 学術雑誌論文 |
| 言語 | 英語 |
| 著作権者 | CopyrightⒸ 2017 by Okayama University Medical School |
| 論文のバージョン | publisher |
| 査読 | 有り |
| PubMed ID | 29276218 |
| JaLCDOI | 10.18926/okadai-bun-kiyou/55578 |
|---|---|
| フルテキストURL | jfl_068_085_100.pdf |
| 著者 | Renoud, Loïc| |
| 出版物タイトル | 岡山大学文学部紀要 |
| 発行日 | 2017-12-22 |
| 巻 | 68巻 |
| 開始ページ | 85 |
| 終了ページ | 100 |
| ISSN | 0285-4864 |
| 言語 | 英語 |
| 論文のバージョン | publisher |
| NAID | 120006370706 |
| JaLCDOI | 10.18926/okadai-bun-kiyou/55577 |
|---|---|
| フルテキストURL | jfl_068_069_083.pdf |
| 著者 | Leonard, Julian| |
| 出版物タイトル | 岡山大学文学部紀要 |
| 発行日 | 2017-12-22 |
| 巻 | 68巻 |
| 開始ページ | 69 |
| 終了ページ | 83 |
| ISSN | 0285-4864 |
| 言語 | 英語 |
| 論文のバージョン | publisher |
| NAID | 120006370705 |
| JaLCDOI | 10.18926/okadai-bun-kiyou/55576 |
|---|---|
| タイトル（別表記） | Beyond the Blue Notebook: The Transcendental Blue in Paul Auster’s Oracle Night（2003） |
| フルテキストURL | jfl_068_055_068.pdf |
| 著者 | 中谷 ひとみ| |
| 出版物タイトル | 岡山大学文学部紀要 |
| 発行日 | 2017-12-22 |
| 巻 | 68巻 |
| 開始ページ | 55 |
| 終了ページ | 68 |
| ISSN | 0285-4864 |
| 言語 | 日本語 |
| 論文のバージョン | publisher |
| NAID | 120006370704 |
| JaLCDOI | 10.18926/okadai-bun-kiyou/55574 |
|---|---|
| タイトル（別表記） | Start of“ The Great Reforms” and Fiscal and Monetary Policy in Imperial Russia |
| フルテキストURL | jfl_068_033_044.pdf |
| 著者 | 吉田 浩| |
| 出版物タイトル | 岡山大学文学部紀要 |
| 発行日 | 2017-12-22 |
| 巻 | 68巻 |
| 開始ページ | 33 |
| 終了ページ | 44 |
| ISSN | 0285-4864 |
| 言語 | 日本語 |
| 論文のバージョン | publisher |
| NAID | 120006370702 |
| JaLCDOI | 10.18926/okadai-bun-kiyou/55572 |
|---|---|
| タイトル（別表記） | A Post-Skinnerian Perspective in Psychology （26）: Old Age as a Radical Behaviorist |
| フルテキストURL | jfl_068_001_017.pdf |
| 著者 | 長谷川 芳典| |
| 抄録 | 本稿は、行動分析学の視点から、高齢者のライフスタイルの構築と終末に関する有用な知見を提供することを目的とする。このテーマは、 （1）スキナーの幸福観の概要と高齢者への適用 （2）選択機会と高齢者の行動的QOL （3）複数の行動のまとまりから構成される「活動」概念に基づいて、より巨視的な観点から高齢者のライフスタイルの構築を考える （4）終末期における不安や恐怖への対処 という4つの観点から総合的に検討する必要があると考えるが、本稿では紙幅の制限により、（2）についてはすでに長谷川（2012, 2013）で論じているのでここでは省略し、また（1）と（4）は概略を述べるにとどめ、新しい概念を取り入れた（3）について重点的に取り上げていくことにしたい。 |
| 出版物タイトル | 岡山大学文学部紀要 |
| 発行日 | 2017-12-22 |
| 巻 | 68巻 |
| 開始ページ | 1 |
| 終了ページ | 17 |
| ISSN | 0285-4864 |
| 関連URL | http://ousar.lib.okayama-u.ac.jp/55220 |
| 言語 | 日本語 |
| 論文のバージョン | publisher |
| NAID | 120006370700 |
| フルテキストURL | K0005600_other1.pdf |
|---|---|
| 著者 | Takeda, Tatsuaki| Yamamoto, Hiromasa| Kanzaki, Hirotaka| Suzawa, Ken| Yoshioka, Takahiro| Tomida, Shuta| Cui, Xiaojiang| Murali, Ramachandran| Namba, Kei| Sato, Hiroki| Torigoe, Hidejiro| Watanabe, Mototsugu| Shien, Kazuhiko| Soh, Junichi| Asano, Hiroaki| Tsukuda, Kazunori| Kitamura, Yoshihisa| Miyoshi, Shinichiro| Sendo, Toshiaki| Toyooka, Shinichi| |
| 備考 | 学位審査副論文| |
| 発行日 | 2017-02-03 |
| 出版物タイトル | PLoS One |
| 巻 | 12巻 |
| 号 | 2号 |
| 出版者 | Public Library of Science |
| 開始ページ | e0171356 |
| ISSN | 1932-6203 |
| 資料タイプ | 学術雑誌論文 |
| 言語 | 英語 |
| OAI-PMH Set | 岡山大学 |
| 著作権者 | https://creativecommons.org/licenses/by-nc-nd/4.0/deed.ja |
| 論文のバージョン | publisher |
| PubMed ID | 28158234 |
| DOI | 10.1371/journal.pone.0171356 |
| Web of Science KeyUT | 000396161700064 |
| 関連URL | isVersionOf https://doi.org/10.1371/journal.pone.0171356 isPartOf http://ousar.lib.okayama-u.ac.jp/55525 |
| フルテキストURL | K0005596_other1.pdf |
|---|---|
| 著者 | Thar Htet San| Fujisawa, Masayoshi| Fushimi, Soichiro| Soe, Lamin| Ngu Wah Min| Yoshimura, Teizo| Ohara, Toshiaki| Myint Myint Yee| Oda, Shinsuke| Matsukawa , Akihiro| |
| キーワード | Breast cancer molecular subtypes Ki-67 expression Myanmar |
| 備考 | 学位審査副論文| |
| 発行日 | 2017-06-25 |
| 出版物タイトル | Asian Pacific journal of cancer prevention |
| 巻 | 18巻 |
| 号 | 6号 |
| 出版者 | Asian Pacific Organization for Cancer Prevention |
| 開始ページ | 1617 |
| 終了ページ | 1621 |
| ISSN | 1513-7368 |
| 資料タイプ | 学術雑誌論文 |
| 言語 | 英語 |
| OAI-PMH Set | 岡山大学 |
| 著作権者 | https://creativecommons.org/licenses/by-nc-nd/4.0/deed.ja |
| 論文のバージョン | publisher |
| PubMed ID | 28670879 |
| DOI | 10.22034/APJCP.2017.18.6.1617 |
| 関連URL | isVersionOf https://doi.org/10.22034/APJCP.2017.18.6.1617 isPartOf http://ousar.lib.okayama-u.ac.jp/55521 |
| フルテキストURL | K0005592_other1.pdf |
|---|---|
| 著者 | Hinamoto, Norikazu| Maeshima, Yohei| Yamasaki, Hiroko| Nasu, Tatsuyo| Saito, Daisuke| Watatani, Hiroyuki| Ujike, Haruyo| Tanabe, Katsuyuki| Masuda, Kana| Arata, Yuka| Sugiyama, Hitoshi| Sato, Yasufumi| Makino, Hirofumi| |
| 備考 | 学位審査副論文| |
| 発行日 | 2014-09-25 |
| 出版物タイトル | Plos One |
| 巻 | 9巻 |
| 号 | 9号 |
| 出版者 | Public Library of Science |
| 開始ページ | e107934 |
| ISSN | 1932-6203 |
| 資料タイプ | 学術雑誌論文 |
| 言語 | 英語 |
| OAI-PMH Set | 岡山大学 |
| 著作権者 | https://creativecommons.org/licenses/by-nc-nd/4.0/ |
| 論文のバージョン | publisher |
| PubMed ID | 25255225 |
| DOI | 10.1371/journal.pone.0107934 |
| Web of Science KeyUT | 000344862300045 |
| 関連URL | isVersionOf https://doi.org/10.1371/journal.pone.0107934 isPartOf http://ousar.lib.okayama-u.ac.jp/55517 |
| フルテキストURL | viruses-09-00371.pdf |
|---|---|
| 著者 | Ogawa, Hirohito| Kajihara, Masahiro| Nao, Naganori| Shigeno, Asako| Fujikura, Daisuke| Hang’ombe, Bernard M.| Mweene, Aaron S.| Mutemwa, Alisheke| Squarre, David| Yamada, Masao| Higashi, Hideaki| Sawa, Hirofumi| Takada, Ayato| |
| キーワード | Eidolon helvum Zambia adenovirus bat |
| 発行日 | 2017-12-04 |
| 出版物タイトル | Viruses |
| 巻 | 9巻 |
| 号 | 12号 |
| 出版者 | MDPI |
| 開始ページ | 371 |
| ISSN | 1999-4915 |
| 資料タイプ | 学術雑誌論文 |
| 言語 | 英語 |
| OAI-PMH Set | 岡山大学 |
| 著作権者 | https://creativecommons.org/licenses/by-nc-nd/4.0/deed.ja |
| 論文のバージョン | publisher |
| PubMed ID | 29207524 |
| DOI | 10.3390/v9120371 |
| 関連URL | isVersionOf https://doi.org/10.3390/v9120371 |
| フルテキストURL | O0004480_abstract_review.pdf O0004480_fulltext.pdf |
|---|---|
| 著者 | Iyasu Getachew Belay| |
| 発行日 | 2017-09-29 |
| 資料タイプ | 学位論文 |
| 学位授与番号 | 乙第4480号 |
| 学位授与年月日 | 2017-09-29 |
| 学位・専攻分野 | 博士（理学） |
| 授与大学 | 岡山大学 |
| 言語 | 英語 |
| フルテキストURL | O0004479_abstract_review.pdf O0004479_fulltext.pdf |
|---|---|
| 著者 | 塔 娜| |
| 発行日 | 2017-09-29 |
| 資料タイプ | 学位論文 |
| 学位授与番号 | 乙第4479号 |
| 学位授与年月日 | 2017-09-29 |
| 学位・専攻分野 | 博士（工学） |
| 授与大学 | 岡山大学 |
| 言語 | 日本語 |
| フルテキストURL | K0005636_abstract_review.pdf.pdf K0005636_fulltext.pdf.pdf |
|---|---|
| 著者 | 呉 揚| |
| 発行日 | 2017-09-29 |
| 資料タイプ | 学位論文 |
| 学位授与番号 | 甲第5636号 |
| 学位授与年月日 | 2017-09-29 |
| 学位・専攻分野 | 博士（文学） |
| 授与大学 | 岡山大学 |
| 言語 | 日本語 |
| フルテキストURL | K0005635_abstract_review.pdf K0005635_summary.pdf K0005635_fulltext.pdf |
|---|---|
| 著者 | 池田 亮| |
| 発行日 | 2017-09-29 |
| 資料タイプ | 学位論文 |
| 学位授与番号 | 甲第5635号 |
| 学位授与年月日 | 2017-09-29 |
| 学位・専攻分野 | 博士（保健学） |
| 授与大学 | 岡山大学 |
| 言語 | 英語 |