岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 エレクトロショックによる事故死の一剖検例 541 544 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 分裂病の研究補遺 第一篇 ビタミンB(1)環流試験に於ける血液瓦斯像に就て 519 540 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 電位測定による抗菌性物質の検定法 第III編 培養液内の発育電位測定による各種抗菌性物質の併用効果検定 511 518 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 電位測定による抗菌性物質の検定法 第II編 嫌気的培養液内の電位測定による抗菌性物質の効果検定 497 510 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 電位測定による抗菌性物質の検定法 第1編 好気的培養液内の電位測定による抗菌性物質の効果検定 479 495 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 アミラーゼ作用に及ぼす単糖類の影響 471 478 EN Mitsuharu Koreeda To investigate what effects would be brought about on starch saccharification by adding certain monosaccharides, we have given 1.0 cc of 1.0% taka-diastase solution to 2.0% starch solution 5.0 cc; moreover. have conducted some saccharification tests by adding 1/1000, 1/100, 1/10 and 1.0 mol solution of glucose, galactose, fructose or mannose, and obtained the following results. (1) The starch saccharification by taka-diastase has been inhibited by an addition of the above-mentioned monosaccharide; especially, most marked in the case of glucose addition, next, galactose, then fructose and mannose, the last being feeblest of them all. After that, we have paid observations on this inhibition degree as well as on the kind of monosaccharides, in comparison with certain structural formulas ascribed to various saccharides. (2) In case 1/1000 mol solution 1.0 cc (the content of each saccharide is 0.18 mg in all cases) is added, in either case where 1.0% taka-diastase solution 1.0 cc or 2.0% starch solution 5.0 cc is concerned (which is same to the bottom), suffer no effect on its saccharification; yet, it has been inhibited considerably in case 1/10 mol solution 1.0 cc (the content of each monosaccharide 18.00 mg) has been added, and has been inhibited to a great degree in case 1.0 mol solution 1.0 cc (the contenl of each monosaccharide 180.00 mg) has been added; on the other hand, in view to an addition of 1/100 mol solution 1.0 cc (the content of each monosaccharide 1.80 mg) it is thought almost out of question on account of very slight difference it has proved against the controls, which is hard to detect any inhibition. At any rate, from 1/1000 to 1.0 mol solution 1.0 cc, i.e., within the range of from 0.18 to 180.00 mg., as the amount of saccharide added becomes greater, the starch saccharification by taka-diastase has proved more and more to suffer inhibition. (3) The content of glucose, galactose, fructose or mannose in each 1.0 mol solution 10 cc being 180.00 mg, corresponds to 1.83 times the amount of the final product of starch hydrolysis (by Liebermann's method), i.e., 98.22mg.; even if such a comparatively considerable amount of monosaccharicde as this is added, there takes place no inhibition whatever for the early stage of starch saccharification. (4) The inhibition of starch saccharification caused by taka-diastase may be, not only owing to glucose, galactose, fructose or mannose, but also owing to various kinds of intermediate products formed in saccharification solution, which have power to thwart starch saccharification caused by taka-diastase. No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 アミラーゼ作用と澱粉並にタカ-ヂアスターゼの濃度との関係 463 470 EN Mitsuharu Koreeda In the saccharification of starch by taka-diastase, in order to clarify to certain extent the correlation that exists between the concentration for both and their final decomposition product, i.e., the quantity of glucose, we have carried out several experiments; the results of which are as follows: (1) Saccharification of starch (2.0, 1.0 and 0.5% solution) by taka-diastase (1.0, 0.5 and 0.1% solution) is brought about very rapidly at the earlier stage of reaction; however, it relaxes rather rapidly, reaching its limit in a certain time. (2) In saccharification of starch, after it has reached its limit, the glucose quantity does not manifest any increase, even if we augment the concentration of taka-diastase solution (ranging from 0.1 to 1.0%), or lengthen the incubation time (to the extent of 72 hours). (3) In case the concentration of starch solution proves fixed (in 0.5, 1.0 or 2.0%), (a) the greater the concentration of taka-diastase solution is, (as to range from 0.1 to 1.0%), the rapider in its saccharification; besides, time needed in order to reach limit, short, and, glucose amount at a certain time (1/2, 3, 6, 12 and 24 hours) proves abundant, but not correspond to the concentration of enzyme solution. (b) When concentration of takadiastase solution is 1.0 and 0.5%, glucose quantity for both at the limit of saccharification proves equal, which has been estimated as about 80% or more of the amount of the final product of starch hydrolysis (by Liebermann's method); moreover, nearly corresponds to concentration of enzyme solution. (c) If the concentration for taka-diastase solution indicates 0.1%, the glucose quantity at the limit of saccharification is to prove approximately 70% or more of the amount of the final product of starch hydrolysis, proving less compared to that shown in 1.0 and 0.5% cases of enzyme solution. (4) When the concentration of taka-diastase solution remains fixed (in 0.1, 0.5 or 1.0%), (a) as the concentration (that ranges from 0.5 to 2.0%) of starch solution proves higher, time needed for it to reach saccharification limit becomes longer. (b) In case the concentration of taka-diastase solution indicates to be 1.0 and 0.5%, glucose amount at the limit of saccharification of 1.0% starch solution proves twice as much; while, that of 2.0% starch solution, compared to that of 0.5% starch solution, proves four times as much. (c) In case the concentration of taka-diastase solution is 0.1%, it is not enough for its saccharification, if starch solution proves 2.0%; while glucose quantity at the limit of saccharification of 1.0% starch solution indicates twice as much of that of 0.5% starch solution, that of 2.0% starch solution does not prove 4 times as much as that of 0.5% starch solution. No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 聽覚による正常人の方向感度 459 462 EN 以上要するに耳軸を含む水平面上の音源に対し,方向感度は前正面附近に於て最良であるとし,前正面より側方に至る間の推移状態を明にし,正常人の数値として示した. No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 聽覚による方向識別に関する研究（其の四）（方向識別機転） 449 458 EN This test was held in a sound-poof room, and the sources of the sound were all of pure tone. The audiometer receivers were connected to a double audiometer. The examinee had an apparent source of sound and the intensity difference and the time difference varied between both ears. The angular displacement of the apparent source was measured in contrast to the pure tone from a loud speaker connected to a beat frequency oscillator and was audible through spaces between the receivers and the ears. It was ascertained that the value of displacement was proportional to the logarithm of the ratio of intensities. (Stewsat's logarithmic law) Stewart's constant is different according to individuals. The constant does not vary with the increasing frequency. The value of the angular displacement was proportional to the difference of the lengths of two sound-ways. The relation of the angular displacement (θ) with the time difference (T) and intensity difference (I) were as follows; θ=f(1)(I)+f(2)(T) Intensity difference must be of extensively great quantity to remove the apparent source. Time difference is the most important factor in actual localization. Intensity have more influence upon localization in acoustically fatigued Person and in patients with deafness. No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 聽覚による方向識別に関する研究（其の三）（難聽者の方向識別） 441 448 EN The source of the sound came from a loud speaker connected to a beat frequency oscillator. The examinees were all patients with hearing defect or deafness. The localisation of the subjective median for the source of the sound in the horizontal plane of their ears were examined and the results were as follows; 1. The monaural localization was unable to be made. 2. They were able to locate the source between 515 d. b. or more from the threshold (of Intensity) by their poorer ear. 3. In ears with equal hearing, the source of the sound was localized accurately in the median. But when the hearing of one ear was better, the source of the sound localized to that one. 4. The position of his apparent median plane in the horizontal plane was shown as the angular displacement. The longer the period of difficult hearing was, the smaller the angular displacement was. (general type of angular displacement) 5. When the threshold was 50-60 d. b. or more (of sensation level), the angular displacement was very great and did not become smaller as the period difficult hearing grew. 6. In patients with neural deafness the range of subjective median was as small as normal hearing, and in a patient with conductive deafness it was very large. 7. After catheterization in the so-called tubotympanic catarrh the range of subjective median become very small, especially during the early stage. This was due to the improvement of conduction mechanism. No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 聽覚による方向識別に関する研究（其の二）（諸條件の影響） 431 439 EN In localizing the source of the sound the following was observed under various conditions in the horizontal plane of the examinee's ears. The range of localization for subjective median, 1. Increased proportionally according to the distance from the source of the sound. 2. Increased by visual stimulus. 3. Increased by auditory fatigue. 4. Decreased when the intense noise was at the background of the source. The sensitivity of localizing sound is separated to two sides, namely the displacement of the apparent source and the range of localization. There has been few reports concerning the latter. Sound localization is produced in the centrum by two auditory nerve excitation. The increased displacement is attributed to unequal nerve excitations, and the increased range of localization to the unclearness of nerve excitation. No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 聽覚による方向識別に関する研究（其の一）（周波数・強さ） 425 429 EN 吾人の聴覚で前正中線と感じるのは一定の拡りを指すのであるが,この範囲は略々解剖学的前正中線附近に正しく局在されている.この範囲は同一周波数の音源では拡りが一定であり,周波数の変化により範囲の大さを異にする.こゝに着眼して周波数による前正中線附近の方向感度を観察し,之が中周波域に於て最も良く,低周波稍々悪く,高周波域に於ては極めて悪い結果を見た.音源の強さに関しては,最小可聴閾より5～15d. b.上昇域に於て略定位可能であるが,多数の人員に対して完全を期する為には最小可聴閾より30d. b.以上の強さを要する事を見た. No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 Anton症状にIsomytalinterviewを試みた一例に就いて 419 424 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 卵膜外リバノール液注入に依る姙娠中絶法 417 418 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 進行性顏面半側萎縮症の2例 411 416 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 Adrenalin-Eosinopenieに関する実験的研究 試験管内血液への添加実験並に骨髄好酸球像の組織学的観察 395 410 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 in vitroにおける大腸菌族の赤痢B群抗元獲得について 391 394 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 鉤虫症鉄代謝に関する研究 第3編 鉤虫症血清注射の貯藏鉄に及ぼす影響 383 390 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 鉤虫症鉄代謝に関する研究 第2編 鉤虫症血清注射の影響並びに網内系の意義 375 381 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 鉤虫症鉄代謝に関する研究 第1編 鉤虫症血清鉄量に関する実験的研究 365 373 EN No potential conflict of interest relevant to this article was reported.

岡山医学会 Acta Medica Okayama 0030-1558 66 3 1954 ホルネル氏症候群を呈せる縱隔竇腫瘍の一例 361 364 EN No potential conflict of interest relevant to this article was reported.