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affil-num=1
en-affil=Graduate School of Environmental and Life Science, Okayama University
kn-affil=‰ªŽR‘åŠw‘åŠw‰@ŠÂ‹«�¶–½‰ÈŠwŒ¤‹†‰È
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dt-pub-year=2020
dt-pub=20200325
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en-title=ƒIƒX‚̃Vƒ‡ƒEƒWƒ‡ƒEƒoƒG‚É‚¨‚¯‚é‰h—{ŠÂ‹«ˆË‘¶“I‚È”D�«�§Œä‚Ì•ªŽq‹@�\
kn-title=Molecular mechanisms of nutrient-dependent regulation of fecundity in Drosophila males
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en-aut-name=MatsukaMirai
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en-affil=Graduate School of Natural Science and Technology, Okayama University
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en-title=The Development Strategies of Poland and the Engine of Polish Economic Growth
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kn-abstract=�@In this paper, authors analyze the changes in the national development strategies of the Polish government since the First World War, the state investment that became the driving force of economic development, the trends and positions of foreign capital, and the changes in the economic environment surrounding the Polish economy. �@In the Second Republic, economic liberalism was a widely supported philosophy in the business world and academic society. However, in order to support very weak economic fundamentals, the government seized the key industry and tried to rebuild the economy. Although foreign capital played a very important role in economic development, Polish people�fs distrust in foreign capital was strong. After the Great Depression, the Central Industrial District�i CUP�j was built under the leadership of the government. However, with the outbreak of World War II, the formation of a strong core industrial base was suspended.<br/>
�@After the Second World War, the government initially aimed to build a Peoples Democracy system based on pluralism. However, when the Cold War sharpened, rapid industrialization and collectivization of agriculture were forced. The Stalinist economic development strategy failed in a few years. Nevertheless, from the end of the 1950s, mining development and industrialization based on the mechanical and chemical industries were carried out again. This industrialization of capital goods sector-oriented production has distorted the people�fs consumer life. However, in the long run, it cannot be denied that the industrialization of that time became the basis for the formation of the wide promising fields of the Polish industry. Since 1970, an open economic strategy has been adopted. Modernization was attempted by introducing licenses from the west. However, new investments did not lead to expansion of export, and cumulative debt increased. During the economic crisis, the�g Solidarity�h movement quickly grew into a national movement, but this did not lead to a fundamental economic system change. Economic reform was also attempted in the 1980s. However, the socialist economic system was unable to adapt to the new global economic system driven by innovation. The inflexible system lost its growth potential.<br/>
�@After the collapse of socialist system in 1989, radical liberalization policies drastically improved economic imbalances, and the economic policy enabled high growth. During the transition period, foreign capital greatly contributed to growth. The automobile industry is a good example. This industry is associated with wide parts production. Many domestic intermediate goods manufacturers increased their orders, and their technical capabilities were rapidly improved through the guidance of foreign capital. In addition, export has improved significantly due to the expansion of production by foreign-affiliated companies. In contrast to the Second Republic, foreign capital played a major role in nurturing domestic industries. By the end of the 1990s, the Russian economy had had no effect on the Polish economy. At the transition period, the Polish industry shifted rapidly to the EU. At the same time, since the manufacturing field was the foundation of Polish growth, the impact of the 2007 global financial crisis wasn�ft serious. Not only that, Poland maintained positive growth in 2008 as most countries in Europe fell into negative Currently, low-wage, low-value-added production based on FDI inflows and abundant labor force is the driving force for growth. In the medium and long term, the Polish economy cannot grow if it stays in the current subcontract production status of industrialized countries. The key to Poland�fs escape from the �gmiddle-income trap�h is how to develop�g new Polish companies�h that produce high-value-added products and services under global competition.
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en-aut-name=TaguchiMasahiro
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en-aut-name=OkazakiTaku
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affil-num=1
en-affil=Graduate School of Humanities and Social Sciences, Okayama University
kn-affil=‰ªŽR‘åŠw‘åŠw‰@ŽÐ‰ï•¶‰»‰ÈŠwŒ¤‹†‰È

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en-title=Œõ‰»Šw“I“à�݉»�iPCI�j‚Ì‹@�\‰ð–¾�C‚¨‚æ‚у}ƒEƒX�‰Šúãó‚Ö‚ÌRNAƒfƒŠƒoƒŠ�[‚ւ̉ž—p
kn-title=The influence of physicochemical parameters on photochemical internalization (PCI) mechanism and application of PCI as a photo-dependent RNA delivery strategy to mouse embryo
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en-aut-name=TET HTUT SOE
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affil-num=1
en-affil=Graduate School of Natural Science and Technology, Okayama University
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en-title=AZIN1 RNA•Ò�W‚ÌŠˆ�«‰»‚Í‘å’°ŠàŠàŠÖ˜A�üˆÛ‰è�×–E‚É‚¨‚¯‚é�Z��”\‚ð‘£�i‚³‚¹‚é�V‚½‚È‹@�˜‚Å‚ ‚é
kn-title=Activation of AZIN1 RNA editing is a novel mechanism that promotes invasive potential of cancer-associated fibroblasts in colorectal cancer
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en-aut-name=TakedaSho
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kn-title=Study on neural mechanisms of temporal expectation on a millisecond to second time scale
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en-aut-name=XuZhihan
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en-title=“î�œ�×–E‚É‚¨‚¯‚é5-HT2A‚¨‚æ‚Ñ5-HT2BŽó—e‘Ì‚ð‰î‚µ‚½ƒZƒ�ƒgƒjƒ“�i5-HT�j‚É‚æ‚éCCN2ŽY�¶‚Ì’²�ß‹@�\
kn-title=Regulatory mechanism of CCN2 production by serotonin (5-HT) via 5-HT2A and 5-HT2B receptors in chondrocytes.
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en-aut-name=HoriAyaka
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affil-num=1
en-affil=Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University
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en-title=EGFR•ÏˆÙ—z�«”ñ�¬�×–E�«”xŠà‚É‚¨‚¯‚éƒIƒVƒ�ƒ‹ƒ`ƒjƒu‘Ï�«‹@�\‚Æ‚µ‚Ä‚ÌAXLŠˆ�«‰»
kn-title=Activation of AXL as a preclinical acquired resistance mechanism against osimertinib treatment in EGFR-mutant non-small cell lung cancer cells
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en-aut-name=NanbaKei
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en-title=MET‘�•�‚¨‚æ‚ÑNRAS‘�•�‚Í‘æ3�¢‘ãEGFR‘jŠQ�܃iƒRƒ`ƒjƒu‚ÌŠl“¾‘Ï�«‹@�\‚Å‚ ‚é
kn-title=MET or NRAS amplification is an acquired resistance mechanism to the third-generation EGFR inhibitor naquotinib
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en-aut-name=NinomiyaKiichiro
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affil-num=1
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en-title=JAK‘jŠQ�܂̃�ƒJƒjƒJƒ‹ƒXƒgƒŒƒX‚É‚æ‚éƒqƒg“î�œ�×–E‚©‚ç‚Ì’`”’•ª‰ð�y‘f”­Œ»—}�§
kn-title=Inhibitory effect of JAK inhibitor on mechanical stress-induced protease expression by human articular chondrocytes
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en-aut-name=MachidaTakahiro
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affil-num=1
en-affil=Graduate School of Medicine, Dentistry and Pharmaceutical Sciences, Okayama University
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en-title=ƒqƒg‘å’°‚ª‚ñ�×–E‚̃xƒ“ƒWƒ‹ƒCƒ\ƒ`ƒIƒVƒAƒl�[ƒg‘Ï�«‹@�\‚ւ̃zƒXƒtƒ@ƒ`ƒWƒ‹ƒCƒmƒVƒg�[ƒ‹ 3-ƒLƒi�[ƒ[‚ÌŠÖ—^
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en-aut-name=LiuXiaoyang
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affil-num=1
en-affil=Graduate School of Environmental and Life Science, Okayama University
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en-title=’†�«Žq‰ñ�Ü‚É‚æ‚é’n‹…ƒ}ƒ“ƒgƒ‹‘JˆÚ‘w�z•¨’†‚Ì�…‘f‚̉ð�Í
kn-title=Hydration mechanism in Earth's mantle transition zone investigated by neutron diffraction
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en-aut-name=PurevjavNarangoo
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affil-num=1
en-affil=Graduate School of Natural Science and Tchnology, Okayama University
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en-title=ˆâ“`Žq”­Œ»‚É‚¨‚¯‚éˆâ“`“I�Û“®‚ɑ΂·‚éŠÉ�Õ‹@�\
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en-aut-name=IshikawaKoji
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affil-num=1
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en-aut-name=EtaniReo
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affil-num=1
en-affil=Graduate School of Health Sciences, Okayama University
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en-title=ƒLƒEƒCƒtƒ‹�[ƒc‰ÊŽÀ‚̃Gƒ`ƒŒƒ“‚É‚æ‚é�¬�n—U“±‚ƒቷ‚É‚æ‚é�¬�n‘£�i‚ÉŠÖ‚·‚é�¶—�Šw“I�E•ªŽq�¶•¨Šw“I‹@�\‰ð�Í
kn-title=Elucidating physiological and molecular mechanisms of ethylene induced ripening and low temperature modulated ripening in kiwifruit
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en-aut-name=WILLIAM OLUBERO ASICHE
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en-title=’·Šú‚ɂ킽‚éCŒ^ŠÌ‰ŠƒEƒCƒ‹ƒX‚ÌRNA•¡�»‚É‚æ‚éCPB2ˆâ“`Žq‚Ì”­Œ»—}�§‹@�\‚̉ð�Í
kn-title=Molecular Mechanism Underlying the Suppression of CPB2 Expression Caused by Persistent Hepatitis C Virus RNA Replication
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en-aut-name=SejimaHiroe
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kn-title=Mechanical Stretch on Human Skin Equivalents Increases the Epidermal Thickness and Develops the Basement Membrane
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ORCID=

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dt-pub-year=2010
dt-pub=20100325
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en-title=Ž–�ÛŠÖ˜A“dˆÊ‚É‚æ‚é�lŠÔ‚ÌŽ‹’®Šo“��‡ƒ�ƒJƒjƒYƒ€‚ÉŠÖ‚·‚錤‹†
kn-title=Study on Human Mechanism of Audiovisual Integration by Event-related Potentials
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en-aut-name=
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kn-aut-name=—›Šï
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en-title=‹…�óƒVƒŠƒJƒtƒBƒ‰�[“Y‰Á‚É‚æ‚é�‡’…—pƒOƒ‰ƒXƒAƒCƒIƒmƒ}�[ƒZƒ“ƒg‚Ì•¨�«‰ü—Ç
kn-title=Mechanical properties of a resin-modified glass-ionomer cement for luting:effect of adding spherical silica filler
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en-aut-name=ELihua
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en-title=‘O�\Žš�x‘Ñ-�œ“î�œŒ‹�‡•”‚É‚¨‚¯‚é�×–E�¶•¨Šw“I“Á’¥�@-ƒ�ƒJƒjƒJƒ‹ƒXƒgƒŒƒX‚É‚æ‚é�×–E�Ú’…‚Æcollagen”­Œ»•Ï‰»‚ÌŒŸ“¢-
kn-title=Mechanical stretch stimulates integrin ƒ¿VƒÀ3 - mediated collagen expression in human anterior cruciate ligament cells
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en-aut-name=TetsunagaTomonori
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dt-pub-year=2009
dt-pub=20090325
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en-title=3-ƒjƒgƒ�ƒvƒ�ƒsƒIƒ“Ž_—U“±�«’P—£ƒ~ƒgƒRƒ“ƒhƒŠƒA–Œ“§‰ß�«‘JˆÚ‚Ì‹@�\‚ÆL-ƒJƒ‹ƒjƒ`ƒ“‚É‚æ‚é‚»‚Ì—}�§
kn-title=Mechanism of 3-nitropropionic acid-induced membrane permeability transition of isolated mitochondria and its suppression by L-carnitine
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kn-subtitle=
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en-aut-name=NishimuraMakoto
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en-aut-mei=Makoto
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affil-num=1
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en-title=ƒ^ƒ“ƒ{ƒRƒIƒ�ƒM‚É‚¨‚¯‚éŒõŽü‘ªŽž‹@�\‚Ì�¶—�Šw“I�E•ªŽq�¶•¨Šw“I‰ð�Í
kn-title=Physiological and molecular analysis of the photoperiodic time-measurement mechanism in the cricket, Modicogryllus siamensis
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kn-subtitle=
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en-aut-name=SakamotoTomoaki
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en-aut-mei=Tomoaki
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affil-num=1
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dt-pub-year=2008
dt-pub=20080930
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en-title=ƒLƒCƒ�ƒVƒ‡ƒEƒWƒ‡ƒEƒoƒG‚Ì匉»‚̃^ƒCƒ~ƒ“ƒO‚ðŒˆ‚ß‚é�¶•¨ƒ^ƒCƒ}�[‚Ì•ªŽq‹@�\
kn-title=Molecular mechanism of a biological timer system that controls pupation timing in Drosophila melanogaster
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en-aut-name=
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kn-aut-name=MOUSTAFA MOHAMEDHEMIDA SARHAN
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ORCID=

affil-num=1
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en-title=ƒ}ƒCƒNƒ�ƒAƒŒƒC‚ð—p‚¢‚½�‚“™�A•¨‚É‚¨‚¯‚é•aŠQ‚¨‚æ‚ъ‹«ƒXƒgƒŒƒX‰ž“š‹@�\‚̉ð–¾
kn-title=Transcriptome analysis of plant defense mechanisms against biotic and abiotic stresses using a microarray
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kn-subtitle=
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en-aut-name=NarusakaMari
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dt-pub-year=2007
dt-pub=20071231
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en-title=HL-60�×–E‚É‚¨‚¯‚éA23187—U“±ƒAƒ|ƒg�[ƒVƒX‚̃�ƒJƒjƒYƒ€�FNADPHƒIƒLƒVƒ_�[ƒ[‚Å‚Í‚È‚­ƒ~ƒgƒRƒ“ƒhƒŠƒA–Œ“§‰ß�«‘JˆÚ‚ւ̈ˑ¶
kn-title=Mechanism of A23187-Induced Apoptosis in HL-60 Cells: Dependency on Mitochondrial Permeability Transition but Not on NADPH Oxidase
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kn-subtitle=
en-abstract=
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en-aut-name=KajitaniNoriko
en-aut-sei=Kajitani
en-aut-mei=Noriko
kn-aut-name=Š�’J“TŽq
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en-title=sks“Ë‘R•ÏˆÙƒ}ƒEƒX‚É‚¨‚¯‚é�¸ŽqŒ`�¬ˆÙ�í‚Ì•ªŽq‹@�\‚ÉŠÖ‚·‚錤‹†
kn-title=Study on the molecular mechanisms of the defect of spermatogenesis in the sks mutant mice
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en-aut-name=AkiyamaKoyo
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en-title=ƒrƒ^ƒ~ƒ“B(12)•â�y‘fŠÖ—^ƒOƒŠƒZƒ��[ƒ‹ƒfƒqƒhƒ‰ƒ^�[ƒ[‚Ì�ÄŠˆ�«‰»ˆöŽq‚Ì“¯’è‚Æ�«Ž¿‚¨‚æ‚Ñ�ì—p‹@�ì
kn-title=Identification, properties, and action mechanism of a reactivating factor for adenosylcobalamin-dependent glycerol dehydratase
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kn-copyright=

en-aut-name=KajiuraHideki
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en-aut-mei=Hideki
kn-aut-name=Š�‰Y‰pŽ÷
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en-title=Ehƒ}ƒEƒX‚É‚¨‚¯‚éŒûŠW—ô‚Ì”­�¶‹@�\‚¨‚æ‚ÑŠÖ˜Aˆâ“`Žq‚ÉŠÖ‚·‚錤‹†
kn-title=Studies on the developmental mechanism and associated genes for cleft palate of Eh mice
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=KatayamaKentaro
en-aut-sei=Katayama
en-aut-mei=Kentaro
kn-aut-name=•ÐŽRŒ’‘¾˜Y
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kn-aut-mei=Œ’‘¾˜Y
aut-affil-num=1
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affil-num=1
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dt-pub-year=2007
dt-pub=20070630
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kn-subject=
en-title=Ž‰–bŠÌƒ}ƒEƒX‚ÌŠÌ�Ø�œŒã‚Ì�Ä�¶�áŠQ‚̃�ƒJƒjƒYƒ€
kn-title=Mechanism of impaired regeneration of fatty liver in mouse partial hepatectomy model
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kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=MurataHiroshi
en-aut-sei=Murata
en-aut-mei=Hiroshi
kn-aut-name=‘º“c�G
kn-aut-sei=‘º“c
kn-aut-mei=�G
aut-affil-num=1
ORCID=

affil-num=1
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kn-affil=‰ªŽR‘åŠw
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dt-received=
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dt-pub-year=2007
dt-pub=20070323
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=‹…�óƒVƒŠƒJƒtƒBƒ‰�[“Y‰Á‚É‚æ‚郌ƒWƒ“ƒ‚ƒfƒBƒtƒ@ƒCƒhƒOƒ‰ƒXƒAƒCƒIƒmƒ}�[ƒZƒ�ƒ“ƒg‚Ì‹@ŠB“I‹­‚³‚̉ü—Ç
kn-title=EFFECTS OF SPHERICAL SIRICA FILLER ADDITION ON IMPROVING THE MECHANICAL CHARACTERISTICS OF REZIN-MODIFIED GLASS-IONOMER CEMENT
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=HatanakaKenji
en-aut-sei=Hatanaka
en-aut-mei=Kenji
kn-aut-name=”¨’†Š£Žu
kn-aut-sei=Ӭՠ
kn-aut-mei=Š£Žu
aut-affil-num=1
ORCID=

affil-num=1
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kn-affil=‰ªŽR‘åŠw
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dt-received=
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dt-accepted=
dt-pub-year=2007
dt-pub=20070323
dt-online=
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kn-subject=
en-title=ƒ‚ƒ‹ƒqƒl’P‰ñ“Š—^ƒ‰ƒbƒg‚É‚¨‚¯‚éƒiƒ�ƒLƒ\ƒ“—U”­�ê�ŠŒ™ˆ«�«ŽŽŒ±‚ɑ΂·‚éƒOƒ‹ƒ^ƒ~ƒ“Ž_Žó—e‘Ì‚ÌŠÖ—^
kn-title=Effect of glutamate receptor antagonists on place aversion induced by naloxone in single-dose morphine-treated rats
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=The neurobiological mechanism underlying the negative motivational component of withdrawal from acute opiate dependence is far from understood. Our objectives were to determine whether the glutamatergic system is involved in the motivational component of morphine withdrawal in acutely dependent rats and such an involvement is associated with dopaminergic neurotransmission. We examined the effects of various kinds of glutamate receptor antagonists on conditioned place aversion (CPA) induced by naloxone-precipitated withdrawal from a single morphine exposure 24 h before. Furthermore, the influence of pretreatment with the dopamine receptor antagonist haloperidol on those effects of glutamate receptor antagonists was also investigated. CPA was attenuated in a dose-dependent manner by all glutamate receptor antagonists examined including the NMDA receptor antagonists (+)-5-methyl-10,11-dihydro-5H-dibenzo[a,d]cyclo-hepten-5,10-imine maleate (MK-801) and phencyclidine hydrochloride (PCP), AMPA receptor antagonist 1-(4-aminophenyl)4-methyl-7,8-methylenedioxy-5H-2,3-benzodiazepine hydrochloride (GYKI 52466), and metabotropic receptor antagonists (+/-)-2-amino-3-phosphonopropionic acid (AP-3) and (+/-)-alpha-methyl-4-carboxyphenylglycine (MCPG). The effects of MK-801, GYKI 52466 and MCPG were blocked by haloperidol.These results suggest that the glutamatergic system involving multiple classes of receptors plays a role in the motivational component of withdrawal from acute morphine dependence, and the function of the glutamatergic system would be closely associated with dopaminergic neurotransmission.
en-copyright=
kn-copyright=

en-aut-name=KawasakiYoichi
en-aut-sei=Kawasaki
en-aut-mei=Yoichi
kn-aut-name=‰Í�è—zˆê
kn-aut-sei=‰Í�è
kn-aut-mei=—zˆê
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
en-keyword=morphine
kn-keyword=morphine
en-keyword=acute dependence
kn-keyword=acute dependence
en-keyword=naloxone
kn-keyword=naloxone
en-keyword=withdrawal
kn-keyword=withdrawal
en-keyword=conditioned place aversion
kn-keyword=conditioned place aversion
en-keyword=glutamate receptor antagonist
kn-keyword=glutamate receptor antagonist
en-keyword=dopamine
kn-keyword=dopamine
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2007
dt-pub=20070323
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ƒwƒŠƒRƒoƒNƒ^�[�Eƒsƒ�ƒŠ”MƒVƒ‡ƒbƒN’`”’60‚Í�A’PŠj‹…�×–E’†‚Å‚ÌTLR2‚ÆMAPƒLƒi�[ƒ[Œo˜H‚ð‰î‚µ�CƒTƒCƒgƒJƒCƒ“IL-8‚Ì•ª”å‚ðˆø‚«‹N‚±‚·
kn-title=Helicobacter pylori heat-shock protein 60 induces interleukin-8 via a toll-like receptor (TLR) 2 and mitogen-activated protein (MAP) kinase pathway in human monocytes
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=Previous reports have indicated that Helicobacter pylori heat-shock protein 60 (H. pylori-HSP60), as an immunodominant antigen, induces interleukin (IL)-8 production in human monocytes. The exact mechanism by which H. pylori-HSP60 induces IL-8 production in monocytes has not been fully elucidated. In the present study, the downstream pathway by which H. pylori-HSP60 induces IL-8 secretion in human monocytic cell lines was investigated. Intact H. pylori, heat-killed H. pylori and H. pylori recombinant HSP60 (rHpHSP60) all induced the secretion of IL-8 and the activation of mitogen-activated protein kinase (MAPK), extracellular signal-regulated kinase (ERK) and p38, but not c-Jun N-terminal kinase (JNK), up to 24 h in NOMO1 cells. The specific inhibitors PD98059 and U0126 (for ERK1/2 signalling) and SB203580 (for p38 MAPK signalling) down-regulated IL-8 secretion from rHpHSP60-treated NOMO1 cells. An anti-Toll-like receptor (TLR)2 antibody or TLR2 small interfering RNA (siRNA) partially inhibited the secretion of IL-8, and anti-TLR2 antibody also suppressed activation of ERK and p38 MAPK in rHpHSP60-treated NOMO1 cells. These reactions were associated with nuclear factor-kappaB (NF-kappaB)-mediated transcriptional activation, since U0126, SB203580 and the anti-TLR2 antibody decreased NF-kappaB activation. Taken together, the results suggest that ERK and p38 MAPK signalling linked to the TLR2 recognition receptor in human monocytes may be an important pathway in H. pylori-HSP60-induced IL-8 secretion.
en-copyright=
kn-copyright=

en-aut-name=ZhaoYing
en-aut-sei=Zhao
en-aut-mei=Ying
kn-aut-name=æâàð
kn-aut-sei=æâ
kn-aut-mei=àð
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
en-keyword=GASTRIC EPITHELIAL-CELLS
kn-keyword=GASTRIC EPITHELIAL-CELLS
en-keyword=CYTOKINE MESSENGER-RNA
kn-keyword=CYTOKINE MESSENGER-RNA
en-keyword=HEAT-SHOCK PROTEINS
kn-keyword=HEAT-SHOCK PROTEINS
en-keyword=KAPPA-B ACTIVATION
kn-keyword=KAPPA-B ACTIVATION
en-keyword=PROINFLAMMATORY CYTOKINES
kn-keyword=PROINFLAMMATORY CYTOKINES
en-keyword=ENDOTHELIAL-CELLS
kn-keyword=ENDOTHELIAL-CELLS
en-keyword=MONGOLIAN GERBILS
kn-keyword=MONGOLIAN GERBILS
en-keyword=LYMPHOID-TISSUE
kn-keyword=LYMPHOID-TISSUE
en-keyword=TLR 2
kn-keyword=TLR 2
en-keyword=MACROPHAGES
kn-keyword=MACROPHAGES
END

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cd-journal=joma
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kn-subject=
en-title=•ÏˆÙFGFR‚R‚Ì�P�í“IƒŠƒ“Ž_‰»‚Í�æ“V�«�¬�l�Ç‚Ì�d—v‚ȈöŽq‚ÅPLCƒÁ‚É‚æ‚éSTAT1‚ÌŠˆ�«‰»‚ð‰î‚µ‚Ä“î�œ‘O‹ì�×–EATDC5‚̃Aƒ|ƒg�[ƒVƒX‚ð—U“±‚·‚é
kn-title=Sustained phosphorylation of mutated FGFR3 is a crucial feature of genetic dwarfism and induces apoptosis in the ATDC5 chondrogenic cell line via PLCƒÁ-activated STAT1
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=The most frequent type of rhizomelic dwarfism, achondroplasia (ACH), is caused by mutations in the fibroblast growth factor receptor 3 (FGFR3) gene. Mutations in FGFR3 result in skeletal dysplasias of variable severity, including mild phenotypic effects in hypochondroplasia (HCH), severe phenotypic effects in thanatophoric dysplasia types I (TDI) and II (TDII), and severe but survivable phenotypic effects in severe achondroplasia with developmental delay and acanthosis nigricans (SADDAN). To explore the molecular mechanisms that result in the different phenotypes, we investigated the kinetics of mutated versions of FGFR3. First, we assayed the phosphorylation states of the mutated FGFR3s and found that the level of phosphorylation in TDI-FGFR3 was lower than in ACH-FGFR3, although the other mutants were phosphorylated according to phenotypic severity. Second, we analyzed the duration of the phosphorylation. TDI-FGFR3 was not highly phosphorylated under ligand-free conditions, but the peak phosphorylation levels of TDI-FGFR3 and ACH-FGFR3 were maintained for 30 min after stimulation with FGF-1. Moreover, ligand-dependent phosphorylation of TDI-FGFR3, but not ACH-FGFR3, lasted for more than 8 h after FGF-1 administration. The other mutant proteins showed sustained phosphorylation independent of ligand presence. Third, we investigated the intracellular localization of the mutant proteins. Immunofluorescence analysis showed accumulations of TDII-FGFR3, SADDAN-FGFR3, and a portion of TDI-FGFR3 in the endoplasmic reticulum (ER). Based on these data, we concluded that sustained phosphorylation of FGFR3 causes chondrodysplasia, and the phenotypic severity depends on the proportion of ER-localized mutant FGFR3. In FGFR3 signaling, the transcription factor, signal transducer and activator of transcription 1 (STAT1) inhibit proliferation and induce apoptosis of chondrocytes. Here we reveal that phospholipase C gamma (PLCgamma) mediates FGFR3-induced STAT1 activation. Both PLCgamma and STAT1 were activated by FGFR3 signaling, but a dominant-negative form of PLCgamma (DN-PLCgamma) remarkably reduced STAT1 phosphorylation. Apoptosis assays revealed that the constitutively active forms of FGFR3 (TDII-FGFR3) and STAT1 (STAT1-C) induce apoptosis of chondrogenic ATDC5 cells via caspase activity. DN-PLCgamma reduced the apoptosis of ATDC5 cells expressing TDII-FGFR3, but over-expression of both DN-PLCgamma and STAT1-C induced apoptosis. Therefore, we conclude that a PLCgamma-STAT1 pathway mediates apoptotic signaling by FGFR3.
en-copyright=
kn-copyright=

en-aut-name=HaradaDaisuke
en-aut-sei=Harada
en-aut-mei=Daisuke
kn-aut-name=Œ´“c‘å•ã
kn-aut-sei=Œ´“c
kn-aut-mei=‘å•ã
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
en-keyword=FGFR3
kn-keyword=FGFR3
en-keyword=Skeletal dysplasia
kn-keyword=Skeletal dysplasia
en-keyword=Phosphorylation
kn-keyword=Phosphorylation
en-keyword=ER localization
kn-keyword=ER localization
en-keyword=STAT1
kn-keyword=STAT1
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2007
dt-pub=20070323
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=‹•ŒŒŒã�ü�ó‘Ì‚É�oŒ»‚·‚é�_Œo‘O‹ì�×–E‚Ì‹NŒ¹
kn-title=Subventricular Zone-Derived Neuroblasts Migrate and Differentiate into Mature Neurons in the Post-Stroke Adult Striatum
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=Recent studies have revealed that the adult mammalian brain has the capacity to regenerate some neurons after various insults. However, the precise mechanism of insult-induced neurogenesis has not been demonstrated. In the normal brain, GFAP-expressing cells in the subventricular zone (SVZ) of the lateral ventricles include a neurogenic cell population that gives rise to olfactory bulb neurons only. Herein, we report evidence that, after a stroke, these cells are capable of producing new neurons outside the olfactory bulbs. SVZ GFAP-expressing cells labeled by a cell-type-specific viral infection method were found to generate neuroblasts that migrated toward the injured striatum after middle cerebral artery occlusion. These neuroblasts in the striatum formed elongated chain-like cell aggregates similar to those in the normal SVZ, and these chains were observed to be closely associated with thin astrocytic processes and blood vessels. Finally, long-term tracing of the green fluorescent-labeled cells with a Cre-loxP system revealed that the SVZ-derived neuroblasts differentiated into mature neurons in the striatum, in which they expressed neuronal-specific nuclear protein and formed synapses with neighboring striatal cells. These results highlight the role of the SVZ in neuronal regeneration after a stroke and its potential as an important therapeutic target for various neurological disorders.
en-copyright=
kn-copyright=

en-aut-name=YamashitaToru
en-aut-sei=Yamashita
en-aut-mei=Toru
kn-aut-name=ŽR‰º“O
kn-aut-sei=ŽR‰º
kn-aut-mei=“O
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
en-keyword=zone
kn-keyword=zone
en-keyword=cerebral ischemia
kn-keyword=cerebral ischemia
en-keyword=migration
kn-keyword=migration
en-keyword=neurogenesis
kn-keyword=neurogenesis
en-keyword=regeneration
kn-keyword=regeneration
en-keyword=striatum
kn-keyword=striatum
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2007
dt-pub=20070323
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ƒ‰ƒbƒgL‚T�Ò�‘�_ŒoŒ‹ãF‚É‚æ‚éƒtƒHƒ‹ƒ}ƒŠƒ“‚ɑ΂·‚éáu’É�s“®•Ï‰»�F�s“®Šw“I‚¨‚æ‚Ñ•ªŽqŠw“IŒŸ“¢
kn-title=Altered Response to Formalin by L5 Spinal Nerve Ligation in Rats: A Behavioral and Molecular Study
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=BACKGROUND: The status of neuropathic pain alters the responsiveness to formalin injection in rats. However, the mechanism by which this alteration occurs is unknown. METHODS: We used immunocytochemistry to examine the expression of brain-derived neurotrophic factor (BDNF) and calcitonin gene-related peptide (CGRP) in the spinal cord of rats with L5 spinal nerve ligation (SNL)-induced neuropathy, and investigated the expression of c-Fos in the spinal cord after injection of formalin in the hindpaw of rats with SNL. RESULTS: Four weeks after SNL, the withdrawal threshold was significantly lower in the SNL group than in the sham-operated (sham) group (n = 12 per group, P < 0.05). In the SNL group, expression of BDNF in the L4 (P < 0.05) and L5 (P < 0.01) superficial dorsal horn was significantly decreased compared to that in the sham group. CGRP protein in the L5 but not in the L4, dorsal horn was significantly decreased compared to that in the sham group (P < 0.01). After formalin injection, spontaneous pain responses in the SNL group were significantly decreased compared to those in the sham group (P < 0.05). Immunolabeling for c-Fos was significantly decreased in the L4 and L5 dorsal horn in the SNL group (P < 0.01). CONCLUSION: Our examination of c-Fos distribution indicates that decreased neuronal activity in the spinal cord in response to inflammatory pain may be important for altering the perception of acute pain. Decreased BDNF expression in response to SNL-induced neuropathy may be involved in this alteration.
en-copyright=
kn-copyright=

en-aut-name=KakuRyuji
en-aut-sei=Kaku
en-aut-mei=Ryuji
kn-aut-name=‰ê—ˆ—²Ž¡
kn-aut-sei=‰ê—ˆ
kn-aut-mei=—²Ž¡
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2007
dt-pub=20070323
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=‚s�×–E�«‘¢ŒŒŠíŽîᇂɂ¨‚¢‚ĉß�è”­Œ»‚ª”F‚ß‚ç‚ê‚éHelios short isoform‚Ì‹@”\‰ð�Í
kn-title=Characterization of the short isoform of Helios overexpressed in patients with T-cell malignancies
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=In an earlier report, we demonstrated overexpression of a short isoform of Helios, Hel-5, which lacks three of four N-terminal zinc fingers, in patients with adult T-cell leukemia/lymphoma. Here, we characterized Hel-5 using immunoprecipitation, and gel shift and luciferase promoter assays, and found that Hel-5 lacks the repressor function observed with a full-length isoform of Helios. Moreover, Hel-5 associates with the full-length isoforms of the Ikaros gene family, Ikaros, Aiolos and Helios, and inhibits their DNA binding activity when present in excess, leading to dominant-negative effects on the full-length isoforms of the Ikaros gene family. Our results suggest a critical role for Helios in the mechanism of leukemogenesis.
en-copyright=
kn-copyright=

en-aut-name=TabayashiTakayuki
en-aut-sei=Tabayashi
en-aut-mei=Takayuki
kn-aut-name=‘½—Ñ�F”V
kn-aut-sei=‘½—Ñ
kn-aut-mei=�F”V
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
en-keyword=Mutation
kn-keyword=Mutation
en-keyword=Carcinogenesis
kn-keyword=Carcinogenesis
en-keyword=Multivariate analysis
kn-keyword=Multivariate analysis
en-keyword=Case-control study
kn-keyword=Case-control study
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2006
dt-pub=20060930
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ƒ`ƒAƒ]ƒŠƒWƒ“—U“±‘Ì‚Í�×–EŽüŠúˆË‘¶�«ƒ�ƒJƒjƒYƒ€‚É‚æ‚è“œ”A•a�«�t�Ç‚ð‰ü‘P‚·‚é
kn-title=Thiazolidinediones Ameliorate Diabetic Nephropathy via Cell Cycle-Dependent Mechanisms
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=OkadaTatsuo
en-aut-sei=Okada
en-aut-mei=Tatsuo
kn-aut-name=‰ª“c’B•v
kn-aut-sei=‰ª“c
kn-aut-mei=’B•v
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2006
dt-pub=20060324
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ƒCƒl‚Ì”­‰è‚¨‚æ‚Ñ—c�A•¨Šú‚É‚¨‚¯‚éƒAƒ‹ƒ~ƒjƒEƒ€‘Ï�«‹@�\‚ÌŒ¤‹†
kn-title=A study of aluminum tolerance mechanisms at germination and seedling stages in rice Oryza sativa L.
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=KikuiSatoshi
en-aut-sei=Kikui
en-aut-mei=Satoshi
kn-aut-name=‹eˆä�¹Žm
kn-aut-sei=‹eˆä
kn-aut-mei=�¹Žm
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2006
dt-pub=20060324
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=�A•¨�×–E‚É‚¨‚¯‚é“œ’`”’Ž¿‚Ì’EƒOƒŠƒRƒVƒ‹‰»‹@�\‚Ɖԕ²ƒAƒŒƒ‹ƒQƒ“‚ÉŒ‹�‡‚·‚éN-ƒOƒŠƒJƒ“‚Ì�\‘¢“Á�«‚ÉŠÖ‚·‚錤‹†
kn-title=Studies on the deglycosylation mechanism of glycoproteins in plant cell and the structural features of N-glycans linked to pollen allergens
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=MaedaMegumi
en-aut-sei=Maeda
en-aut-mei=Megumi
kn-aut-name=‘O“cŒb
kn-aut-sei=‘O“c
kn-aut-mei=Œb
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2006
dt-pub=20060324
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ƒTƒ‹ƒ‚ƒlƒ‰•Ú–Ñ‘Š•ÏˆÙ‚É‚¨‚¯‚é1‘Šƒtƒ‰ƒWƒFƒŠƒ“ˆâ“`Žq‚ÌFljA‚É‚æ‚é—}�§‹@�\
kn-title=Molecular mechanism of FljA-mediated repression of the phase-1 flagellin gene in flagellar phase variation of Salmonella enterica serovar Typhimurium
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=YamamotoShoji
en-aut-sei=Yamamoto
en-aut-mei=Shoji
kn-aut-name=ŽR–{�ÍŽ¡
kn-aut-sei=ŽR–{
kn-aut-mei=�ÍŽ¡
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2006
dt-pub=20060324
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=’°ŠÔ–Œ“®–¬’ï�RŒŒŠÇ‚É‚¨‚¯‚éƒqƒXƒ^ƒ~ƒ“‚É‚æ‚錌ŠÇ”½‰ž‚Ì‹@�˜‚ÉŠÖ‚·‚é–ò—�Šw“IŒ¤‹†
kn-title=Pharmacological study on mechanisms underlying vascular responses to histamine in mesenteric resistance arteries.
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=‹à�g‰Ô
kn-aut-sei=‹à
kn-aut-mei=�g‰Ô
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2006
dt-pub=20060324
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=�¬–EŒ^ƒOƒ‹ƒ^ƒ~ƒ“Ž_—A‘—‘̂̃gƒ|ƒ�ƒW�[‚ƃ^�[ƒQƒbƒeƒBƒ“ƒO‚ÉŠÖ‚·‚錤‹†
kn-title=Topology and Targeting Mechanism of Vesicular Glutamate Transporter
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=“A�æ‹ž
kn-aut-sei=“A
kn-aut-mei=�拞
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2006
dt-pub=20060324
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=‚Q‚g�|‹y‚Ñ‚S‚g�|ƒAƒ[ƒsƒ“‚Ì�‡�¬�C�¶�¬‹@�\‚Ɖ»Šw“I‹““®‚ÉŠÖ‚·‚錤‹†
kn-title=Efficient Synthesis, Formation Mechanism and Chemical Behavior of 2H- and 4H-Azepines
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=CHRISTOPHERERNEST JOHN CORDONIER
kn-aut-sei=CHRISTOPHER
kn-aut-mei=ERNEST JOHN CORDONIER
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2006
dt-pub=20060324
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=•êŽq–ƉuŠ°—e‚Í�§Œä�«T�×–E‚ð‰î‚µ‚ÄŽq•êŠÔ�œ�‘ˆÚ�A‚Ì�¬�Ñ‚ð‰ü‘P‚·‚é
kn-title=Fetal Tolerance to Maternal Antigens Improves the Outcome of Allogeneic Bone Marrow Transplantation by a CD4+CD25+ T cell-Dependent Mechanism
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=MatsuokaKen-ichi
en-aut-sei=Matsuoka
en-aut-mei=Ken-ichi
kn-aut-name=�¼‰ªŒ«Žs
kn-aut-sei=�¼‰ª
kn-aut-mei=Œ«Žs
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2004
dt-pub=20040325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ƒ‚ƒ‹ƒqƒl’P‰ñ“Š—^ƒ‰ƒbƒg‚É‚¨‚¯‚éƒiƒ�ƒLƒ\ƒ“—U”­�ðŒ�•t‚¯�ê�ŠŒ™ˆ«�s“®‹@�˜‚ÉŠÖ‚·‚é�_Œo‰»Šw‹y‚Ñ�_Œo‰ð–UŠw“IŒ¤‹†
kn-title=Neurochemical and neuroanatomic study on the mechanism erlying naloxone-induced conditioned place aversion in rats undergoing a single morphine exposure
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=JinChunyu
en-aut-sei=Jin
en-aut-mei=Chunyu
kn-aut-name=‹à�t‹Ê
kn-aut-sei=‹à
kn-aut-mei=�t‹Ê
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2005
dt-pub=20050325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ƒŠƒ“ƒNŒ^—ò‹ì“®‹@ŠBƒVƒXƒeƒ€‚Ì”ñ�üŒ`�§Œä
kn-title=Nonlinear Control of Underactuated Linked Mechanical Systems
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=HenmiTomohiro
en-aut-sei=Henmi
en-aut-mei=Tomohiro
kn-aut-name=ˆíŒ©’m�O
kn-aut-sei=ˆíŒ©
kn-aut-mei=’m�O
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2004
dt-pub=20040325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=Ž•‰ÈƒCƒ“ƒvƒ‰ƒ“ƒg‚̈À’è�«‚¨‚æ‚ÑŒ`�óƒAƒZƒXƒ�ƒ“ƒg‚Ì‚½‚ß‚Ì‹@ŠBƒ‚ƒrƒŠƒeƒB‹Z–@‚ÉŠÖ‚·‚錤‹†
kn-title=Mechanical Mobility Technique for Stability and Geometry Assessment of Dental Implant
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=WijayaSastra Kusuma
en-aut-sei=Wijaya
en-aut-mei=Sastra Kusuma
kn-aut-name=ƒEƒBƒWƒƒƒ„ƒTƒXƒgƒ‰ ƒNƒXƒ}
kn-aut-sei=ƒEƒBƒWƒƒƒ„
kn-aut-mei=ƒTƒXƒgƒ‰ ƒNƒXƒ}
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2004
dt-pub=20040630
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ˆ³”——Í‚É‚æ‚é•IŠÖ�ß’E‰P�œ�Ü‚Ì”­�¶‹@�˜‚Ì—ÍŠw“I‰ð�Í
kn-title=Biomechanical analysis of the mechanism of elbow fracture-dislocations by compression force
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=WakeHirofumi
en-aut-sei=Wake
en-aut-mei=Hirofumi
kn-aut-name=˜aŸ†”Ž•¶
kn-aut-sei=˜aŸ†
kn-aut-mei=”Ž•¶
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2005
dt-pub=20050325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=CHO�×–E‚É‚¨‚¯‚é˜Z‰¿ƒNƒ�ƒ€—U“±‚̃Aƒ|ƒg�[ƒVƒX‹@�\‚ɂ̓Zƒ‰ƒ~ƒh‚ªŠÖ—^‚·‚é
kn-title=Involvement of Ceramide in the Mechanism of Cr(VI)-induced Apoptosis of CHO Cells
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=MuranakaShikibu
en-aut-sei=Muranaka
en-aut-mei=Shikibu
kn-aut-name=‘º’†Ž®•”
kn-aut-sei=Լՠ
kn-aut-mei=Ž®•”
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2005
dt-pub=20050630
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=15-ƒfƒIƒLƒV-12,14-ƒvƒ�ƒXƒ^ƒOƒ‰ƒ“ƒWƒ“J2‚É‚æ‚é“î�œ“÷Žî�×–E‚Ì�¬’·—}�§‹@�˜‚É‚¨‚¯‚éBax/Bcl-xL‚Æp21”­Œ»•Ï‰»
kn-title=Suppression of chondrosarcoma cells by 15-deoxy-Delta 12,14-prostaglandin J2 is associated with altered expression of Bax/Bcl-xL and p21
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=We previously reported that 15-deoxy-Delta(12,14)-prostaglandin J(2) (15d-PGJ(2)), the most potent agonist for peroxisome proliferator-activated receptor gamma (PPAR gamma), induces apoptosis of human chondrosarcoma cell line OUMS-27. The current study aimed to explore the mechanism of 15d-PGJ(2)-induced apoptosis and inhibition of cell proliferation in OUMS-27 cells. The preliminary results of cDNA microarray analysis showed the down-regulation of anti-apoptotic Bcl-xL and up-regulation of pro-apoptotic Bax in the process of 15d-PGJ(2)-induced apoptosis. These changes were further confirmed at mRNA and protein levels by RT-PCR and Western blot analysis, respectively. Among cyclin-dependent kinase inhibitors, p21 was induced and up-regulated by 15d-PGJ(2), but p16 and p27 were not changed, suggesting that the involvement of p21 in inhibition of cell proliferation. Activation of caspase-3 by 15d-PGJ(2) was partly, but not completely, blocked by PPAR gamma antagonist (GW9662) suggesting the 15d-PGJ(2) exerted its effect by PPAR gamma-dependent and -independent pathways. Interestingly, immunohistochemical study on human chondrosarcoma samples revealed that Bcl-xL is frequently expressed by tumor cells. The results of the current study suggest that the potential ability of 15d-PGJ(2) in regulation of cell cycle and inhibition of Bcl-xL expression might be beneficial in the development of novel pharmacological agents for chondrosarcoma.
en-copyright=
kn-copyright=

en-aut-name=ShenZheng-Nan
en-aut-sei=Shen
en-aut-mei=Zheng-Nan
kn-aut-name=�\�³’j
kn-aut-sei=�\
kn-aut-mei=�³’j
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
en-keyword=Chondrosarcoma
kn-keyword=Chondrosarcoma
en-keyword=Apoptosis
kn-keyword=Apoptosis
en-keyword=PPARƒÁ
kn-keyword=PPARƒÁ
en-keyword=15d-PGJ2
kn-keyword=15d-PGJ2
en-keyword=Bcl-xL
kn-keyword=Bcl-xL
en-keyword=p21
kn-keyword=p21
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2004
dt-pub=20040930
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=Osteoclastogenesis Inhibitory Factor/Osteoprotegerin‚Í—ÍŠw“I•‰‰×ŒyŒ¸‚É‚æ‚é�œŒ¸�­‚ð—}�§‚µ‚½
kn-title=Osteoclastogenesis Inhibitory Factor/Osteoprotegerin Reduced Bone Loss Induced by Mechanical Unloading
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=IchinoseYojiro
en-aut-sei=Ichinose
en-aut-mei=Yojiro
kn-aut-name=ˆêƒmûP—mŽŸ˜Y
kn-aut-sei=ˆêƒmûP
kn-aut-mei=—mŽŸ˜Y
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1999
dt-pub=19990325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ˆÙ•û“I‚È‚ç‚Ñ‚É—ÊŽq‹ÉŒÀ‚Ì’´“`“±‘Ì‚É‚¨‚¯‚é‰Q�\‘¢
kn-title=Vortex structure in anisotropic and quantum-limit superconductors
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=Electronic structure of a single vortex (or vortex core structure) in type-II superconductors is theoretically discussed in the present thesis. Low-lying excited states in the superconductors due to the vortex, i.e., "vortex bound states," are examined in detail on the basis of numerical calculations. Two points are focused on: the effect of superconducting gap anisotropy on a vortex (Chapter 2) and the property of a vortex in quantum-limit situation (Chapter 3). The anisotropy of a superconducting energy gap has substantial effects on the structure of the vortex bound states. The local density of states around a vortex is calculated in a clean superconductor with the gap anisotropy within the framework of the quasiclassical theory of superconductivity. A characteristic structure of the local density of states, observed experimentally in the layered hexagonal superconductor 2H-NbSe(2) by scanning tunneling microscopy (STM), is well reproduced by assuming an anisotropic s-wave gap. The local density of states (or the bound states) around a vortex in superconductors with gap anisotropy is interpreted in terms of quasiparticle trajectories to facilitate an understanding of the rich electronic structure observed in STM experiments. This reveals not only a rich internal electronic structure associated with a vortex core, but also unique ability of the STM spectroscopy. The quantum limit means that the superconducting coherence length is small in the limit, i.e., it is comparable to the atomic length order. Focusing on quantum-limit behavior, fundamental structure of a vortex is studied by self-consistently solving the Bogoliubov-de Gennes equation. The discreteness of the energy levels of the vortex bound states is crucial for the vortex structure in the quantum limit. The following are revealed by the study of the quantum limit. The vortex core radius shrinks monotonically up to an atomic-scale length on
lowering the temperature T, and the shrinkage stops to saturate at a lower T. The pair potential, supercurrent, and local density of states around the vortex exhibit Friedel-like oscillations. The local density of states inside a vortex core generally has particle-hole asymmetry induced by the existence of the vortex itself. Some essential properties of general vortices which are concealed within the conventional non-quantum-limit analysis can be extracted by the quantum-limit analysis. On the basis of the inherent particle-hole asymmetry inside vortex cores, it is discussed in this thesis that electric charging of a vortex core is originated from the Friedel oscillation of the Bogoliubov wave functions around the vortex (Chapter 4) . This mechanism of the vortex core charging is independent of the slope in the normal-state density of states at the Fermi level. The temperature dependence of the vortex core charge is also presented. It is expected that by using STM, information on the vortex core charging is extracted through a relation between the vortex core charge and the vortex bound states.
en-copyright=
kn-copyright=

en-aut-name=HayashiNobuhiko
en-aut-sei=Hayashi
en-aut-mei=Nobuhiko
kn-aut-name=—Ñ�L•F
kn-aut-sei=—Ñ
kn-aut-mei=�L•F
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1998
dt-pub=19980930
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=Œõ‰»ŠwŒnII D1 ’`”’Ž¿‘O‹ì‘̃Jƒ‹ƒ{ƒLƒVƒ‹––’[ƒvƒ�ƒZƒVƒ“ƒO�y‘f‚̊”FŽ¯‹@�\
kn-title=Mechanism of substrate-recognition- by a protease in the carboxy1- terminal processing of precursor D1 protein in photosystem II.
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=“cŒû•x”üŽq
kn-aut-sei=“cŒû
kn-aut-mei=•x”üŽq
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1995
dt-pub=19950325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ƒRƒIƒ�ƒM�¶�BŠíŠ¯‚Ì—ÍŠwŠˆ“®‚É‚¨‚¯‚é�_Œo•ª”å�×–E‚Ì‹@”\“I–ðŠ„
kn-title=Functional role of neurosecretory cells in the mechanical activity of the reproductive organs in the crlcket (Grllus bimaculatus)
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=’•k
kn-aut-sei=’Â
kn-aut-mei=•k
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1996
dt-pub=19960325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ƒRƒIƒ�ƒMŽ€‚É‚Ü‚Ë�s“®‚Ì�_Œo‹@�\
kn-title=Neural mechanism of thanatosis in the cricket
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=�¼–ì�_Žj
kn-aut-sei=�¼–ì
kn-aut-mei=�_Žj
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1998
dt-pub=19980325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=—YƒRƒIƒ�ƒM‚ÌŒð”ö�s“®—}�§‹@�\
kn-title=Inhibitory mechanism of mating behavior in the male cricket
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=�¼–{�K‹v
kn-aut-sei=�¼–{
kn-aut-mei=�K‹v
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
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article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1997
dt-pub=19970325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=—ÞŽ—‘Š“¯(ƒzƒ�ƒIƒ�ƒKƒX)‹y‚Ñ”ñ‘Š“¯(ƒmƒ“ƒzƒ‚ƒ�ƒKƒX)DNAŠÔ‚Ì“¯•ûŒü”½•œ”z—ñ‚Ɉˑ¶‚·‚錇Ž¸Œ`�¬‚Ì•ªŽq‹@�\:‘å’°‹Ûˆê–{�½DNAŒ‹�‡’`”’Ž¿SSB‚Ì‹@”\
kn-title=Mechanism for Deletion Formation Depending on Direct Repeats between Homeologous Sequences and between Nonhomologous Sequences:Involvement of the Escherichia coli Single- Stranded DNA-Binding Protein(SSB)
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=ŒüŒ´—²•¶
kn-aut-sei=ΟΫ
kn-aut-mei=—²•¶
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
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cd-vols=
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article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1996
dt-pub=19960930
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=�‚“™�A•¨‚É‚¨‚¯‚éŒõ�‡�¬Ž_‘f”­�¶Œn’`”’Ž¿‚Ì�W�‡‰ß’ö:ƒAƒZƒ“ƒuƒŠ�[•”ˆÊ‹y‚ÑŒ‹�‡ƒ�ƒJƒjƒYƒ€‚ÌŒ¤‹†
kn-title=Assembly of photosynthetic oxygen- evolving complex subunits in higher plants:sites of assembly and mechanism of binding
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=HashimotoAkiko
en-aut-sei=Hashimoto
en-aut-mei=Akiko
kn-aut-name=‹´–{�»Žq
kn-aut-sei=‹´–{
kn-aut-mei=�»Žq
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
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article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1997
dt-pub=19970325
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en-article=
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kn-subject=
en-title=•ú�o‹@�˜‚ÌŠÏ“_‚©‚ç‚È‚³‚ꂽ�Ò’Å‹y‚Ñ–³�Ò’Å“®•¨�_Œo‹Ø�Ú�‡•”‚É‚¨‚¯‚鎩”­�«‹y‚Ñ—U”­�«�_Œo“`’B•¨Ž¿•ú�o‚ÉŠÖ‚·‚錤‹†
kn-title=Studies of spontaneous and evoked neurotransmitter release at verte- brate and invertebrate neuromuscular junctions from the viewpoint of the mechanism for release
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=Šì‘½�O
kn-aut-sei=Šì‘½
kn-aut-mei=�O
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1995
dt-pub=19950325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=Ž_‘fŠÖ˜Aƒ‰ƒWƒJƒ‹Ží‚É‚æ‚é”|—{�S‹Ø�×–E�áŠQ:�áŠQ‚̃�ƒJƒjƒYƒ€‚È‚ç‚Ñ‚É�×–E“à–hŒä‹@�\‚̉ð–¾
kn-title=Oxygen derived free radical-induced cultured cardiac myocyte damages: evaluation of the mechanisms of the injuries and the cellular defense system
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=�¼’J—F�d
kn-aut-sei=�¼’J
kn-aut-mei=—F�d
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2002
dt-pub=20020325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=’†‰·�«�¬�‡‰h—{“SŽ_‰»�׋ÛOKM-9Š”‚É‚¨‚¯‚é�¬�‡‰h—{“I‘ãŽÓ‹@�\‰ð–¾‚ÉŠÖ‚·‚錤‹†
kn-title=Studies on the Mechanism of Mixotrophic Metabolism in a Mesophilic, Mixotrophic Iron-oxidizing Bacterium Strain OKM-9
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=ˆä�ãŒh•v
kn-aut-sei=ˆä�ã
kn-aut-mei=Œh•v
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
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start-ver=1.4
cd-journal=joma
no-vol=
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article-no=
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end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2002
dt-pub=20020325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ŠÒŒ³/Ž_‰»‚É‚æ‚é�‚Œ´Žq‰¿ƒIƒLƒ\“Sƒ|ƒ‹ƒt¨ƒŠƒ“‚Ì�¶�¬‹@�\ -ƒXƒgƒbƒvƒgƒtƒ��[–@‚É‚æ‚錤‹†-
kn-title=Formation Mechanism of High-Valent Oxo-Iron Porphyrins Induced by the Reduction/Oxidation of Iron(III)-Porphyrin -Studies by Stopped-Flow Techniques-
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=TapanKumar Saha
kn-aut-sei=Tapan
kn-aut-mei=Kumar Saha
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2000
dt-pub=20000325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=�‚“™�A•¨‚É‚¨‚¯‚郀ƒVƒ‰�[ƒQ‚Æ—L‹@Ž_‚É‚æ‚éƒAƒ‹ƒ~ƒjƒEƒ€–³“ʼn»‹@�\
kn-title=Aluminum Detoxification Mechanism by Organic Acids and Mucilage in Higher Plants
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=êt‹Å•ô
kn-aut-sei=êt
kn-aut-mei=‹Å•ô
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1999
dt-pub=19990325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=�¬”ž‚É‚¨‚¯‚éƒAƒ‹ƒ~ƒjƒEƒ€—U“±“SƒNƒ�ƒ�ƒVƒX‚Ì‹@�\‚ɂ‚¢‚Ä
kn-title=Mechanisms of Aluminum-Induced Iron Chlorosis in Wheat (Triticum aestivum )
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=’£åYŒ‰
kn-aut-sei=’£
kn-aut-mei=åYŒ‰
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

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cd-journal=joma
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dt-received=
dt-revised=
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dt-pub-year=2000
dt-pub=20000930
dt-online=
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en-title=ƒnƒCƒ“ƒc�¬‘ÌŒ`�¬‚É‚æ‚é�ÔŒŒ‹…•ÏŒ`”\’ቺ‚Ì‹@�˜‚ÉŠÖ‚·‚錤‹†
kn-title=On the Mechanism of Loss of Deformability in Human Erythrocytes due to Heinz Body Formation : A Flow EPR Study
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en-title=ƒ}ƒNƒ�ƒt§�[ƒW‚Ì•‰‰×“dˆÙ•¨æÃ�H‹@�\‚ÉŠÖ‚·‚錤‹†
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en-title=In vitro‚É‚¨‚¯‚銴�ì‹Ç�ŠƒŠƒmƒp�ß�×–E‚Ì•W“I�×–E‚ɑ΂·‚é�ì—p‹@�ì‚ɂ‚¢‚Ä
kn-title=Action mechanism in vitro of sensitized regional lymph node cells on target cell
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en-title=The mechanism of human basophil activation through the low affinity IgG receptor (Fc ƒÁ R‡U) : Analysis of calcium mobilization using a new flow cytometric method
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en-aut-name=SuwakiToshimitsu
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dt-pub-year=1988
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en-title=ƒAƒ“ƒ‚ƒjƒA‰ð“Å‹@�\‚É‚¨‚¯‚é”A‘f,ƒOƒ‹ƒ^ƒ~ƒ“�‡�¬Œn‚Ì“®“IŠÖ˜A:Œ€�NJ̉Šƒ‰ƒbƒg‚Å‚Ì15NH4Cl‚ð—p‚¢‚½’Ç�ÕŽÀŒ±
kn-title=Dynamic Relationship Between Urea And Glutamine Synthesis in the Mechanism of Ammonia Detoxication : A Tracer Study Using 15NH4Cl in Fulminant Hepatic Failure Rats
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affil-num=1
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dt-pub-year=1991
dt-pub=19910328
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en-title=“V‘RŒ^ƒqƒgŽîᇉ󎀈öŽq‚Æ“V‘RŒ^ƒqƒgƒCƒ“ƒ^�[ƒtªƒ�ƒ“-ƒ¿‚Ì•¹—p‚É‚æ‚é�RŽîᇌø‰Ê‚Ì�×–EŽüŠú‚©‚ç‚Ý‚½ƒ�ƒJƒjƒYƒ€‚ÌŒŸ“¢
kn-title=Mechanism of the Combined Antitumor Effect of Natural Human Tumor Necrosis Factor-ƒ¿ and Natural Human Interferon-ƒ¿ on Cell Cycle Progression
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en-aut-name=
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kn-aut-name=Žº‰ë•F
kn-aut-sei=Žº
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affil-num=1
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kn-affil=‰ªŽR‘åŠw
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cd-journal=joma
no-vol=
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article-no=
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dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1992
dt-pub=19920331
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kn-subject=
en-title=”xˆÚ�AŒã‚̌ċz•s‘S‚ɑ΂·‚é Veno-venous ECMO ‚ÌŽÀŒ±“IŒ¤‹†
kn-title=Experimental Study on Veno-venous Extracorporeal Membrane Oxygenation for Respiratory Failure after Lung Transplantation
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=Extracorporeal Membrane Oxygenation (ECMO) has been adopted as a means of strong respiratory support. In lung transplantation, reimplantation response is still a serious problem. It causes severe respiratory failure which is refractory to mechanical ventilation in some cases. The purpose of this study was to evaluate the effects of veno-venous ECMO after lung transplantation using a canine autotransplantation model. The autotransplantation model was created by keeping the left lung in a warm ischemic state for 2 h. After reperfusion, the right pulmonary artery was ligated. The following two groups were studied: Group 1, Control group, (no ECMO group) (n = 6). After reperfusion, both lungs were ventilated without ECMO. Group 2, ECMO group (n = 7). After reperfusion, veno-venous ECMO support was introduced with reduction of mechanical ventilation. In the no ECMO group, four of the animals died within 210 min after reperfusion. In the ECMO group, two of the animals died of severe pulmonary edema. Data of blood gas analyses (PaO2, PaCO2, and SvO2) after reperfusion were significantly better in the ECMO group, whereas there were no significant differences in both shunt fraction and pulmonary vascular resistance index. In this model with severe pulmonary edema induced by warm ischemia, veno-venous ECMO contributed to the improvement of hypoxemia and hypercapnia, but did not improve pulmonary hemodynamics.
en-copyright=
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en-aut-name=IchibaShingo
en-aut-sei=Ichiba
en-aut-mei=Shingo
kn-aut-name=Žs�ê�WŒá
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aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
en-keyword=extracorporeal membrance oxygenation(ECMO)
kn-keyword=extracorporeal membrance oxygenation(ECMO)
en-keyword=warm ischemia, reimplantation
kn-keyword=warm ischemia, reimplantation
en-keyword=lung transplantation
kn-keyword=lung transplantation
en-keyword=pulmonary edema
kn-keyword=pulmonary edema
en-keyword=veno-venous ECMO
kn-keyword=veno-venous ECMO
END

start-ver=1.4
cd-journal=joma
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article-no=
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dt-received=
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dt-accepted=
dt-pub-year=2002
dt-pub=20020325
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en-title=‹�–Ô–Œ�F‘f�ã”ç�×–E‚ÌTIMP-1ŽY�¶‚ÍŽüŠú“I�L“W‚É”½‰ž‚µ‚Ä‘�‰Á‚·‚é
kn-title=TIMP-1 Production by Bovine Retinal Pigment Epithelial Cells Increases in Response to Cyclic Mechanical Stretch
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kn-subtitle=
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en-aut-name=
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en-aut-mei=
kn-aut-name=ŽRŒûŽ÷ˆê˜Y
kn-aut-sei=ŽRŒû
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ORCID=

affil-num=1
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cd-journal=joma
no-vol=
cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=2000
dt-pub=20001231
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en-title=ŽüŠú“I�L“W—Í‚É‚æ‚é�l‹­–Œ�üˆÛ‰è�×–E‚ÌTIMP-1ŽY�¶Œ¸�­
kn-title=TIMP-1 production by human scleral fibroblasts decreases in response to cyclic mechanical stretching
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en-aut-name=
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kn-aut-name=ŽR‰ª�º�G
kn-aut-sei=ŽR‰ª
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affil-num=1
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dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1979
dt-pub=19790331
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kn-subject=
en-title=ŠÌŽ¾Š³‚É‚¨‚¯‚éŠÌ�y‘f‚Ì—V�o‹@�˜
kn-title=The mechanism of the release of hepatic enzymes in various liver diseases.
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kn-title=MECHANICAL STRENGTH OF INTRAMEDULLARY PINNING AND TRANSFRAGMENTAL KIRSCHNER WIRE FIXATION FOR COLLES' FRACTURES
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kn-title=A Novel Gene(Oculomedin)Induced by Mechanical Stretching in Human Trabecular Cells of the Eye
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en-title=•ª—£‚µ‚½ƒ‰ƒcƒg”ì–ž�×–E‚©‚ç‚̃¿-ƒLƒ‚ƒgƒŠƒvƒVƒ“‚É‚æ‚éƒqƒXƒ^ƒ~ƒ“—V—£‚Ì‹@�\
kn-title=Mechanism of histamine release by alpha-cymotrypsin from isolated rat mast cells
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en-title=ƒtƒbƒgƒVƒ‡ƒbƒN‚É‚æ‚éƒ}ƒEƒX”]“àƒqƒXƒ^ƒ~ƒ“‘ãŽÓ‰ñ“]‚̘´�i‚É‚¨‚¯‚éƒIƒsƒIƒCƒh‚¨‚æ‚Ñ”ñƒIƒsƒIƒCƒh‹@�\‚ÌŠÖ—^
kn-title=Involvement of opioid and non-opioid mechanisms in footshock-induced enhancement of brain histamine turnover in mice
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en-title=ŠàŠ³ŽÒ‚ɉ—‚¯‚éŽè�p�N�P‚É‚æ‚éNK�×–EŠˆ�«‚Ì—}�§‚Æ‚»‚Ì—}�§‹@�\‚ÉŠÖ‚·‚錤‹†
kn-title=Suppression of Natural Killer Cell Activity by Surgical Stress in Cancer Patients and the Underlying Mechanisms
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en-aut-name=
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kn-aut-name=‹gŒ´‹vŽi
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dt-pub-year=2001
dt-pub=20011231
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en-title=ƒOƒ‰ƒ€‰A�«ž…‹Û¥—z�«‹…‹Û‹Nˆö�«‚Ì�¶‘Ì–hŒä‹@�\‚É‚¨‚¯‚éƒTƒCƒgƒJƒCƒ“”½‰ž‚Ì�·ˆÙ:’`”’•ª‰ð�y‘f‘jŠQ�Ügabexate mesilateŽg—p‚̈Ӌ`
kn-title=Differential cytokine response in host defense mechanisms triggered by gram-negative and gram-positive bacteria: roles of gabexate mesilate, a synthetic protease inhibitor
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kn-subtitle=
en-abstract=
kn-abstract=
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en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=Šâ“¡�_“T
kn-aut-sei=Šâ“¡
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affil-num=1
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start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
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article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1999
dt-pub=19990331
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ŽÀŒ±“I�‚ŒŒˆ³ƒ‚ƒfƒ‹‚É‚¨‚¯‚é‰AŒsŒŒŠÇ‚Ì�\‘¢•Ï‰»‚ɂ‚¢‚Ä
kn-title=The penis is not protected--in hypertension there are vascular changes in the penis which are similar to those in other vascular beds
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=In hypertension, small arteries in a variety of vascular beds undergo structural changes that increase resistance. To assess whether there are differential structural changes in the penis that accompany hypertension, we began with determining structurally-based vascular resistance properties in penile and hindlimb vascular beds of adult spontaneously hypertensive rats (SHR) and Sprague-Dawley (SD) rats. In anesthetized SHR, the penile and hindlimb vasculature were isolated and perfused, maximum dilation was induced, and a flow-pressure assessment and alpha 1-adrenoceptor agonist concentration-response curves were generated. Both the baseline and maximum constrictor responses were similar in the two beds of each strain, and overall the maximum structurally-based vascular resistance in SHR was higher than in SD rats. Our data suggests that the penile vasculature is not protected from the structural changes that take place in the other vascular beds in hypertension. There does not appear to be an underlying functional control mechanism that protects the penile vasculature from structural changes that may have a negative impact on penile blood flow.
en-copyright=
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en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=‰ª•”�_“T
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aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
en-keyword=erectile dysfunction
kn-keyword=erectile dysfunction
en-keyword=hypertension
kn-keyword=hypertension
en-keyword=structural change
kn-keyword=structural change
en-keyword=penile vasculature
kn-keyword=penile vasculature
en-keyword=spontaneously hypertensive rats
kn-keyword=spontaneously hypertensive rats
END

start-ver=1.4
cd-journal=joma
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article-no=
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dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1998
dt-pub=19980325
dt-online=
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kn-article=
en-subject=
kn-subject=
en-title=ŽüŠú“I�L“W—͂ɑ΂·‚éƒEƒV‚Ì�üˆÛ’Œ‘Ñ�×–E‚ÌTIMP-1,MMP-2•ª”å
kn-title=Bovine Trabecular Cells Produce TIMP-1 and MMP-'2 in Response to Mechanical Stretching
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kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=‰ª“c—R”üŽq
kn-aut-sei=‰ª“c
kn-aut-mei=—R”üŽq
aut-affil-num=1
ORCID=

affil-num=1
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kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
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article-no=
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end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1990
dt-pub=19900930
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=MLR—}�§ˆöŽq‚ðŽY�¶‚·‚éƒqƒgT�×–EƒnƒCƒuƒŠƒh�[ƒ}‚ÌŽ÷—§:ƒhƒi�[“ÁˆÙ“I—AŒŒ‚Ì�ì—pƒ�ƒJƒjƒYƒ€‚Æ‚ÌŠÖŒW
kn-title=Establishment of a Human T Cell Hybridoma Producing an MLR Suppressing Factor:Relationship to the Mechanism of Action of Donor-Specific Transfusion (DST)
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kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
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en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=‰Hˆä�²ŽÀ
kn-aut-sei=‰Hˆä�²
kn-aut-mei=ŽÀ
aut-affil-num=1
ORCID=

affil-num=1
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kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
no-vol=
cd-vols=
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article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1991
dt-pub=19911231
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=—r�…’†ƒhƒpƒ~ƒ“ŽY�¶‹@�\
kn-title=The Production Mechanism of Amniotic Fluid Dopamine in Rats
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kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=ˆä�ã˜a‘¥
kn-aut-sei=ˆä�ã
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aut-affil-num=1
ORCID=

affil-num=1
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kn-affil=‰ªŽR‘åŠw
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dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1992
dt-pub=19920331
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=•Ó‰�Œn”­�ì�d�Ï�ó‘Ô‚ÆŠC”n�_Œo�×–E’E—Ž‚Ì�¬ˆö‚ɂ‚¢‚Ä: deep Prepyriform cortex ‚Ö‚ÌŠÔŒ‡“I“d‹CŽhŒƒ‚É‚æ‚郉ƒbƒg�d�σ‚ƒfƒ‹‚ð—p‚¢‚½ŽÀŒ±“IŒ¤‹†
kn-title=Mechanisms in the development of limbic status epilepticus and hippocampal neuron loss : an experimental study in a model of status epileticus induced by kindling-like electrical stimulation of the deep prepyriform cortex in rats
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kn-subtitle=
en-abstract=
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en-copyright=
kn-copyright=

en-aut-name=
en-aut-sei=
en-aut-mei=
kn-aut-name=ˆä�ãŒõ‘¾˜Y
kn-aut-sei=ˆä�ã
kn-aut-mei=Œõ‘¾˜Y
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
END

start-ver=1.4
cd-journal=joma
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cd-vols=
no-issue=
article-no=
start-page=
end-page=
dt-received=
dt-revised=
dt-accepted=
dt-pub-year=1996
dt-pub=19960325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=�V‚µ‚¢ƒAƒvƒ��[ƒ`‚É‚æ‚郉ƒbƒg�S‹ØƒXƒ‰ƒCƒX‚Ì‹@ŠB“I–³•‰‰×�ó‘Ô‚Å‚Ì�S‹ØŽ_‘f�Á”ï—Ê
kn-title=Myocardial oxygen consumption of mechanically unloaded contraction of rat ventricular slices by a new approach
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=YasuharaShingo
en-aut-sei=Yasuhara
en-aut-mei=Shingo
kn-aut-name=ˆÀŒ´�LŒá
kn-aut-sei=ˆÀŒ´
kn-aut-mei=�LŒá
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
en-keyword=rat ventricular slices
kn-keyword=rat ventricular slices
en-keyword=Myocardial oxygen consumption
kn-keyword=Myocardial oxygen consumption
en-keyword=mechanically unloaded contraction
kn-keyword=mechanically unloaded contraction
END

start-ver=1.4
cd-journal=joma
no-vol=
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article-no=
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dt-received=
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dt-accepted=
dt-pub-year=2001
dt-pub=20010325
dt-online=
en-article=
kn-article=
en-subject=
kn-subject=
en-title=ŽüŠú“I�L“W—Í‚É‚æ‚é–Ô–Œ�×–E‚ÌTIMP-1, TIMP-2‚ÌŽY�¶‘�‰Á
kn-title=Retinal Cells Produce TIMP-1 and TIMP-2 in Response to Cyclic Mechanical Stretching
en-subtitle=
kn-subtitle=
en-abstract=
kn-abstract=
en-copyright=
kn-copyright=

en-aut-name=NanbaMasako
en-aut-sei=Nanba
en-aut-mei=Masako
kn-aut-name=“ï”g‰ëŽq
kn-aut-sei=“ï”g
kn-aut-mei=‰ëŽq
aut-affil-num=1
ORCID=

affil-num=1
en-affil=
kn-affil=‰ªŽR‘åŠw
en-keyword=TIMP-1
kn-keyword=TIMP-1
en-keyword=TIMP-2
kn-keyword=TIMP-2
en-keyword=Cyclic Mechanical Stretching
kn-keyword=Cyclic Mechanical Stretching
END