ID | 69190 |
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suppl.docx
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Author |
Orimo, Kenta
Department of Neurology, Graduate School of Medicine, The University of Tokyo
Matsukawa, Takashi
Department of Neurology, Graduate School of Medicine, The University of Tokyo
Mitsutake, Akihiko
Department of Neurology, Graduate School of Medicine, The University of Tokyo
Cho, Takusei
Department of Neurology, Graduate School of Medicine, The University of Tokyo
Naruse, Hiroya
Department of Neurology, Graduate School of Medicine, The University of Tokyo
Sakiyama, Yoshio
Division of Neurology, First Department of Integrated Medicine, Saitama Medical Center, Jichi Medical University
Sumi, Kensho
Department of Neurology, Mitsui Memorial Hospital
Uchio, Naohiro
Department of Neurology, Mitsui Memorial Hospital
Satake, Akane
Department of Neurology, Fuefuki Central Hospital
Takiyama, Yoshihisa
Department of Neurology, Fuefuki Central Hospital
Matsushita, Takuya
Department of Neurology, Kochi Medical School, Kochi University
Omae, Yosuke
Genome Medical Science Project, National Center for Global Health and Medicine
Kawai, Yosuke
Genome Medical Science Project, National Center for Global Health and Medicine
Tokunaga, Katsushi
Genome Medical Science Project, National Center for Global Health and Medicine
Mitsui, Jun
Department of Precision Medicine Neurology, Graduate School of Medicine, The University of Tokyo
Ishiura, Hiroyuki
Department of Neurology, Graduate School of Medicine, The University of Tokyo
Tsuji, Shoji
Department of Neurology, Graduate School of Medicine, The University of Tokyo
Toda, Tatsushi
Department of Neurology, Graduate School of Medicine, The University of Tokyo
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Abstract | Biallelic loss-of-function variants in CLCN2 lead to CLCN2-related leukoencephalopathy (CC2L), also called leukoencephalopathy with ataxia (LKPAT). CC2L is characterized clinically by a spectrum of clinical presentations including childhood- to adult-onset mild ataxia, spasticity, cognitive decline, and vision loss as well as typical MRI findings of symmetrical high signal intensities on the DWIs/T2WIs of the middle cerebellar peduncles (MCPs). We searched for pathogenic variants of CLCN2 in a case series of undiagnosed leukoencephalopathy accompanied by MCP signs, which led to the identification of four Japanese patients with CC2L. All the patients carried at least one allele of c.61dupC (p.Leu21Profs*27) in CLCN2, including compound heterozygosity with either the novel pathogenic variant c.983 + 2 T > A or the previously reported pathogenic variant c.1828C > T (p.Arg610*). Of note, all the four previously reported cases from Japan also harbored c.61dupC, and no reports of this variant have been documented from outside Japan. The allele frequency of c.61dupC in the Japanese population is 0.002152, raising the possibility of a relatively high prevalence of CC2L in Japan. Patients in this study developed symptoms after the age of 30, and demonstrated neurological signs including cerebellar ataxia, pyramidal signs, and mild cognitive impairment, consistent with previous reports. One male patient had two children, supporting preserved fertility, and another patient had calcifications in the cerebral and cerebellar surfaces. These findings provide valuable insights into the broader clinical and genetic spectra of CC2L in the Japanese population, and emphasize the importance of considering this disease in the differential diagnoses of leukoencephalopathy with MCP signs.
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Keywords | Leukodystrophy
CC2L
CLCN2
MCP sign
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Published Date | 2025-05
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Publication Title |
Journal of the Neurological Sciences
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Volume | volume472
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Publisher | Elsevier BV
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Start Page | 123486
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ISSN | 0022-510X
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NCID | AA00703265
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Content Type |
Journal Article
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language |
English
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OAI-PMH Set |
岡山大学
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Copyright Holders | © 2025 The Authors.
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File Version | publisher
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PubMed ID | |
DOI | |
Web of Science KeyUT | |
Related Url | isVersionOf https://doi.org/10.1016/j.jns.2025.123486
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License | http://creativecommons.org/licenses/by/4.0/
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助成情報 |
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22ek0109493:
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23ek0109617:
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( 国立研究開発法人日本医療研究開発機構 / Japan Agency for Medical Research and Development )
22K07533:
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( 文部科学省 / Ministry of Education )
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