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ID 13443
Eprint ID
13443
著者
木村 真士 岡山大学大学院医歯学総合研究所 消化器・腫瘍外科学
羽井佐 実 岡山大学大学院医歯学総合研究所 消化器・腫瘍外科学
高岡 宗徳 岡山大学大学院医歯学総合研究所 消化器・腫瘍外科学
伴 秀利 岡山大学大学院医歯学総合研究所 消化器・腫瘍外科学
上塚 大一 岡山大学大学院医歯学総合研究所 消化器・腫瘍外科学
大川 尚臣 岡山大学大学院医歯学総合研究所 消化器・腫瘍外科学
河島 留一 岡山大学大学院医歯学総合研究所 消化器・腫瘍外科学
白川 靖博 岡山大学大学院医歯学総合研究所 消化器・腫瘍外科学
繁光 薫 岡山大学大学院医歯学総合研究所 消化器・腫瘍外科学
山辻 知樹 岡山大学大学院医歯学総合研究所 消化器・腫瘍外科学
猶本 良夫 岡山大学大学院医歯学総合研究所 消化器・腫瘍外科学
田中 紀章 岡山大学大学院医歯学総合研究所 消化器・腫瘍外科学
キーワード
TNF
IFN-α
Apoptosis
NF-κB
Fas
備考
特別依頼原稿
発行日
2005-01-31
出版物タイトル
岡山医学会雑誌
出版物タイトル(別表記)
Journal of Okayama Medical Association
116巻
3号
出版者
岡山医学会
出版者(別表記)
Okayama Medical Association
開始ページ
221
終了ページ
228
ISSN
0030-1558
NCID
AN00032489
資料タイプ
学術雑誌論文
オフィシャル URL
https://www.jstage.jst.go.jp/article/joma1947/116/3/116_3_221/_article/-char/ja/
関連URL
http://www.okayama-u.ac.jp/user/oma/
参考文献
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2) Muro M, Naomoto Y and Orita K: Mechanism of the combined antitumor effect of natural human tumor necrosis factor-α and natural human interferon-α on cell cycle progression. Jpn J Cancer Res (1991) 82, 118-126.
3) Satomoto K, Haisa M, Naomoto Y, Tanaka N, Orita K: Cyclin A and Cdk2 kinase activity are suppressed by combined treatment with tumor necrosis factor-alpha and interferonalpha. Biochem Biophys Res Commun (1995) 213, 1115-1121.
4) Yasuoka Y, Naomoto Y, Yamatsuji T, Takaoka M, Kimura M, Uetsuka H, Matsubara N, Fujiwara T, Gunduz M, Tanaka N, Haisa M: Combination of tumor necrosis factor alpha and interferon alpha induces apoptotic cell death through a c-myc-dependent pathway in p53 mutant H226br non-small-cell lung cancer cell line. Exp Cell Res (2001) 271, 214-222.
5) Hsu H, Shu H-B, Pan M-G and Goeddel DV: TRADD-TRAF2 and TRADD-FADD interactions define two distinct TNF receptor 1 signal transduction pathways. Cell (1996) 84, 299-308.
6) Boldin MP, Goncharov TM, Goltsev YV and Wallach D: Involvement of MACH, a novel MORT1/FADD-interacting protease, in Fas/APO-1-and TNF receptor-induced cell death. Cell (1996) 85, 803-815.
7) Muzio M, Chinnaiyan AM, Kischkel FC, O'Rourke K, Shevchenko A, Ni J, Scaffidi C, Bretz JD, Zhang M, Gentz R, Mann M, Krammer PH, Peter ME and Dixit VM: FLICE, A Novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death-inducing signaling complex. Cell (1996) 85, 817-827.
8) Malinin NL, Boldin MP, Kovalenko AV and Wallach D: MAP3K-related kinase involved in NF-KB induction by TNF, CD95 and IL-1. Nature (1997) 385, 540-544.
9) Didonato JA, Hayakawa M, Rothwarf DM, Zandi E and Karin M: A cytokine-responsive IκB kinase that activates the transcription factor NF-κB. Nature (1997) 388, 548-554.
10) Zandi E, Rothwarf DM, Delhase M, Hayakawa M and Karin M: The IκB kinase complex (IKK) contains two kinase subunits, IKKα and IKKβ, necessary for IκB phosphorylation and NFκB activation. Cell (1997) 91, 243-252.
11) Baeuerle PA and Baltimore D: IκB: A specific inhibitor of the NF-κB transcription factor. Science (1988) 242, 540-546.
12) Traenckner EB-M, Pahl HL, Henkel T, Schmidt KN, Wilk S and Baeuerle PA: Phosphorylation of human IκB-α on serines 32 and 36 controls IκB-α proteolysis and NF-κB activation in response to diverse stimuli. EMBO J (1995) 14, 2876-2883.
13) Morishita R, Sugimoto T, Aoki M, Kida I, Tomita N, Moriguchi A, Maeda K, Sawa Y, Kaneda Y, Higaki J and Ogihara T: In vivo transfection of cis element “decoy” against nuclear factor-κB binding site prevents myocardial infarction. Nat Med (1997) 3, 894-899.
14) Hosoya T, Takeuchi H, Kanesaka Y, Yamakawa H, Miyano-Kurosaki N, Takai K, Yamamoto N and Takaku H: Sequencespecific inhibition of a transcription factor by circular dumbbell DNA oligonucleotides. FEBS Lett (1999) 461, 136-140.
15) Kaneda Y, Uchida T, Kim J, Ishiura M and Okada Y: The improved efficient method for introducing macromolecules into cells using HVJ (Sendai Virus) liposomes with gangliosides. Exp Cell Res (1987) 173, 56-69.
16) Saeki Y, Matsumoto N, Nakano Y, Mori M, Awai K and Kaneda Y: Development and characterization of cationic liposomes conjugated with HVJ (Sendai Virus): Reciprocal effect of cationic lipid for in vitro and in vivo. Gene transfer. Hum Gene Therapy (1997) 8, 2133-2141.
17) Chan H, Bartos DP and Owen-Schaub LB: Activationdependent transcriptional regulation of the human Fas promoter requires NF-κB p50-p65 recruitment. Mol Cell Biol (1999) 19, 2098-2108.
18) Ouaaz F, Li M and Beg AA: A critical role for the Rel A subunit of nuclear factor-κB in regulation of multiple immune-response genes and in Fas-induced cell death. J Exp Med (1999) 189, 999-1004.
19) Lin B, Williams-Skipp C, Tao Y, Schleicher MS, Cano LL, Duke RC and Scheinman RI: NF-κB functions as both a proapoptotic and antiapoptotic regulatory factor within a single cell type. Cell Death Differ (1999) 6, 570-582.
20) Garban HJ, Bonavida B: Nitric oxide sensitizes ovarian tumor cells to Fas-induced apoptosis. Gynecol Oncol (1999) 73, 257-264.
21) Garban HJ, Bonavida B: Nitric oxide inhibits the transcription repressor Yin-Yang 1 binding activity at the silencer region of the Fas promoter: A pivotal role for nitric oxide in the up-regulation of Fas gene expression in human tumor cells. J Immunol (2001) 167, 75-81.
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言語
Japanese
OAI-PMH Set
岡山大学
著作権者
Copyright© 岡山医学会
論文のバージョン
publisher
査読
有り
Sort Key
2
Eprints Journal Name
joma